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Abbas K. Rizi

Publications and source records attributed to Abbas K. Rizi.

9 recordsLinked to original sources

What is emergence, after all?

The term emergence is increasingly used across scientific disciplines to describe phenomena that arise from interactions among a system's components but cannot be readily inferred by examining those components in isolation. While often invoked to explain higher-level behaviors, such as flocking, synchronization, or collective intelligence, the term is frequently used without precision, sometimes giving rise to ambiguity or even mystique. In this perspective paper, we clarify the scientific meaning of emergence as a measurable and physically grounded phenomenon. Through concrete examples, such as temperature, magnetism, and herd immunity in social networks, we review how collective behavior can arise from local interactions that are constrained by global boundaries. By refining the concept of emergence, we gain a clearer and more grounded understanding of complex systems. Our goal is to show that emergence, when properly framed, offers not mysticism but insight.

physics.soc-ph

Homophily Within and Across Groups

Homophily -- the tendency of individuals to interact with similar others -- shapes how networks form and function. Yet existing approaches typically collapse homophily to a single scale, either one parameter for the whole network or one per community, thereby detaching it from other structural features. Here, we introduce a maximum-entropy random graph model that moves beyond these limits, capturing homophily across all social scales in the network, with parameters for each group size. The framework decomposes homophily into within- and across-group contributions, recovering the stochastic block model as a special case. As an exponential-family model, it fits empirical data and enables inference of group-level variation of homophily that aggregate metrics miss. The group-dependence of homophily substantially impacts network percolation thresholds, altering predictions for epidemic spread, information diffusion, and the effectiveness of interventions. Ignoring such heterogeneity risks systematically misjudging connectivity and dynamics in complex systems.

physics.soc-ph

Strength and weakness of disease-induced herd immunity in networks

When a fraction of a population becomes immune to an infectious disease, the population-wide infection risk decreases nonlinearly due to collective protection, known as herd immunity. Some studies based on mean-field models suggest that natural infection in a heterogeneous population may induce herd immunity more efficiently than homogeneous immunization. However, we theoretically show that this is not necessarily the case when the population is modeled as a network instead of using the mean-field approach. We identify two competing mechanisms driving disease-induced herd immunity in networks: the biased distribution of immunity toward socially active individuals enhances herd immunity, while the topological localization of immune individuals weakens it. The effect of localization is stronger in networks embedded in a low-dimensional space, which can make disease-induced immunity less effective than random immunization. Our results highlight the role of networks in shaping herd immunity and call for a careful examination of model predictions that inform public health policies.

physics.soc-ph

Story Arcs, Success and Diffusion of Turkish TV Dramas

Turkish TV dramas, renowned for their extensive episodes and captivating romantic narratives, have gained global popularity, leading to the central question of this study: what drives the virality of these series based on their scripts and emotional story arcs? This study explores the global popularity of Turkish TV dramas, examining their narrative structures and emotional arcs to understand their widespread appeal. It delves into the cultural and emotional factors that resonate with international audiences, highlighting Türkiye's significant role in the global entertainment landscape. Through a combination of sentiment analysis and narrative examination, the research offers insights into how these dramas create emotional engagement and cultural connectivity, contributing to Türkiye's growing influence in global media.

physics.soc-ph

Effectiveness of Contact Tracing on Networks with Cliques

Contact tracing, the practice of isolating individuals who have been in contact with infected individuals, is an effective and practical way of containing disease spread. Here, we show that this strategy is particularly effective in the presence of social groups: Once the disease enters a group, contact tracing not only cuts direct infection paths but can also pre-emptively quarantine group members such that it will cut indirect spreading routes. We show these results by using a deliberately stylized model that allows us to isolate the effect of contact tracing within the clique structure of the network where the contagion is spreading. This will enable us to derive mean-field approximations and epidemic thresholds to demonstrate the efficiency of contact tracing in social networks with small groups. This analysis shows that contact tracing in networks with groups is more efficient the larger the groups are. We show how these results can be understood by approximating the combination of disease spreading and contact tracing with a complex contagion process where every failed infection attempt will lead to a lower infection probability in the following attempts. Our results illustrate how contact tracing in real-world settings can be more efficient than predicted by models that treat the system as fully mixed or the network structure as locally tree-like.

physics.soc-ph

Directed Percolation in Random Temporal Network Models with Heterogeneities

The event graph representation of temporal networks suggests that the connectivity of temporal structures can be mapped to a directed percolation problem. However, similar to percolation theory on static networks, this mapping is valid under the approximation that the structure and interaction dynamics of the temporal network are determined by its local properties, and otherwise, it is maximally random. We challenge these conditions and demonstrate the robustness of this mapping in case of more complicated systems. We systematically analyze random and regular network topologies and heterogeneous link-activation processes driven by bursty renewal or self-exciting processes using numerical simulation and finite-size scaling methods. We find that the critical percolation exponents characterizing the temporal network are not sensitive to many structural and dynamical network heterogeneities, while they recover known scaling exponents characterizing directed percolation on low dimensional lattices. While it is not possible to demonstrate the validity of this mapping for all temporal network models, our results establish the first batch of evidence supporting the robustness of the scaling relationships in the limited-time reachability of temporal networks.

physics.soc-ph

Directed Percolation in Temporal Networks

Connectivity and reachability on temporal networks, which can describe the spreading of a disease, decimation of information or the accessibility of a public transport system over time, have been among the main contemporary areas of study in complex systems for the last decade. However, while isotropic percolation theory successfully describes connectivity in static networks, a similar description has not been yet developed for temporal networks. Here address this problem and formalize a mapping of the concept of temporal network reachability to percolation theory. We show that the limited-waiting-time reachability, a generic notion of constrained connectivity in temporal networks, displays directed percolation phase transition in connectivity. Consequently, the critical percolation properties of spreading processes on temporal networks can be estimated by a set of known exponents characterising the directed percolation universality class. This result is robust across a diverse set of temporal network models with different temporal and topological heterogeneities, while by using our methodology we uncover similar reachability phase transitions in real temporal networks too. These findings open up an avenue to apply theory, concepts and methodology from the well-developed directed percolation literature to temporal networks.

physics.soc-ph

Epidemic Spreading and Digital Contact Tracing: Effects of Heterogeneous Mixing and Quarantine Failures

Contact tracing via digital tracking applications installed on mobile phones is an important tool for controlling epidemic spreading. Its effectivity can be quantified by modifying the standard methodology for analyzing percolation and connectivity of contact networks. We apply this framework to networks with varying degree distributions, numbers of application users, and probabilities of quarantine failures. Further, we study structured populations with homophily and heterophily and the possibility of degree-targeted application distribution. Our results are based on a combination of explicit simulations and mean-field analysis. They indicate that there can be major differences in the epidemic size and epidemic probabilities which are equivalent in the normal SIR processes. Further, degree heterogeneity is seen to be especially important for the epidemic threshold but not as much for the epidemic size. The probability that tracing leads to quarantines is not as important as the application adoption rate. Finally, both strong homophily and especially heterophily with regard to application adoption can be detrimental. Overall, epidemic dynamics are very sensitive to all of the parameter values we tested out, which makes the problem of estimating the effect of digital contact tracing an inherently multidimensional problem.

physics.soc-ph

Herd Immunity and Epidemic Size in Networks with Vaccination Homophily

We study how the herd immunity threshold and the expected epidemic size depend on homophily with respect to vaccine adoption. We find that the presence of homophily considerably increases the critical vaccine coverage needed for herd immunity and that strong homophily can push the threshold entirely out of reach. The epidemic size monotonically increases as a function of homophily strength for a perfect vaccine, while it is maximized at a nontrivial level of homophily when the vaccine efficacy is limited. Our results highlight the importance of vaccination homophily in epidemic modeling.

physics.soc-ph