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Andrea Tosin

Publications and source records attributed to Andrea Tosin.

At least 19 recordsLinked to original sources

Derivation and quasi-invariant asymptotics of phenotype-structured integro-differential models

Building upon kinetic theory approaches for mass-varying multi-agent systems, we develop a modelling framework for phenotype-structured populations that makes it possible to bridge individual-level mechanisms with population-scale evolutionary dynamics. We start by formulating a stochastic agent-based model, which describes the dynamics of single population members undergoing proliferation, death, and phenotype changes. Then, we formally derive the corresponding mesoscopic model, which consists of an integro-differential equation for the distribution of population members over the space of phenotypes, where phenotype changes are modelled via an integral kernel. Finally, considering a quasi-invariant regime of small but frequent phenotype changes, we rigorously derive a non-local Fokker-Planck-type equation counterpart of this model, wherein phenotype changes are taken into account by an advection-diffusion term. The theoretical results obtained are illustrated through a sample of results of numerical simulations.

math.AP

Homogeneous Boltzmann-type equations on dense graphs

In kinetic theory, interactions between particles are typically assumed to be "all-to-all", meaning that any pair of randomly selected particles may, in principle, interact. This assumption originates from the theory of colliding gas molecules; however, it may be less appropriate for describing other forms of interaction, such as social interactions. These are more naturally characterised as "some-to-some", reflecting the existence of preferential connections between agents. In this paper, we consider homogeneous Boltzmann-type equations on finite graphs that model such networks of preferential interactions, and we rigorously derive their dense graph limit as the number of agents tends to infinity. We also investigate the long-time behaviour of the limiting equation in the case of linear pairwise interactions, characterising the emergent equilibrium distributions and relating them to their counterparts in the classical "all-to-all" setting.

math-ph

Homogeneous Boltzmann-type equations on graphs: A framework for modelling networked social interactions

Homogeneous Boltzmann-type equations are an established tool for modelling interacting multi-agent systems in sociophysics by means of the principles of statistical mechanics and kinetic theory. A customary implicit assumption is that interactions are "all-to-all", meaning that every pair of randomly sampled agents may potentially interact. However, this legacy of classical kinetic theory, developed for collisions among gas molecules, may not be equally applicable to social interactions, which are often influenced by preferential connections between agents. In this paper, we discuss ongoing research on incorporating graph structures into homogeneous Boltzmann-type equations, thereby accounting for the "some-to-some" nature of social interactions.

physics.soc-ph

Heterogeneously structured compartmental models of epidemiological systems: from individual-level processes to population-scale dynamics

We develop a general modelling framework for compartmental epidemiological systems structured by continuous variables which are linked to the levels of expression of compartment-specific traits. We start by formulating an individual-based model that describes the dynamics of single individuals in terms of stochastic processes. Then we formally derive: (i) the mesoscopic counterpart of this model, which is formulated as a system of integro-differential equations for the distributions of individuals over the structuring-variable domains of the different compartments; (ii) the corresponding macroscopic model, which takes the form of a system of ordinary differential equations for the fractions of individuals in the different compartments and the mean levels of expression of the traits represented by the structuring variables. We employ a reduced version of the macroscopic model to obtain a general formula for the basic reproduction number, $\mathcal{R}_0$, in terms of key parameters and functions of the underlying microscopic model, so as to illustrate how such a modelling framework makes it possible to draw connections between fundamental individual-level processes and population-scale dynamics. Finally we apply the modelling framework to case studies based on classical compartmental epidemiological systems, for each of which we report on Monte Carlo simulations of the individual-based model as well as on analytical results and numerical solutions of the macroscopic model.

q-bio.PE

Linear inelastic kinetic equations modelling the spread of fake news and its interplay with personal awareness

In this paper, we introduce a kinetic model which describes a learning process leading individuals to build personal awareness about fake news. Next, we embed the results of this model into another kinetic model, which describes the popularity gained by news on social media conditioned to the reliability of the disseminated information. Both models are formulated in terms of linear inelastic Boltzmann-type equations, of which we investigate the main analytical properties - existence and uniqueness of solutions, trend to equilibrium, identification of the equilibrium distributions - by employing extensively Fourier methods for kinetic equations. We also provide evidence of the analytical results by means of Monte Carlo numerical simulations.

physics.soc-ph

Probabilistic modeling of car traffic accidents

We introduce a counting process to model the random occurrence in time of car traffic accidents, taking into account some aspects of the self-excitation typical of this phenomenon. By combining methods from probability and differential equations, we study this stochastic process in terms of its statistical moments and large-time trend. Moreover, we derive analytically the probability density functions of the times of occurrence of traffic accidents and of the time elapsing between two consecutive accidents. Finally, we demonstrate the suitability of our modelling approach by means of numerical simulations, which address also a comparison with real data of weekly trends of traffic accidents.

physics.soc-ph

Essentials of the kinetic theory of multi-agent systems

In this paper, we present a critical collection of essential mathematical tools and techniques for the analysis of Boltzmann-type kinetic equations, which in recent years have established themselves as a flexible and powerful paradigm to model interacting multi-agent systems. We consider, in particular, scalar equations implementing linear symmetric interaction rules, for which we develop the theory of well-posedness, trend to equilibrium, and Fokker-Planck asymptotics by relying extensively on Fourier methods. We also outline the basics of Monte Carlo algorithms for the numerical solution of such equations. Finally, we elaborate the theory further for Boltzmann-type equations on graphs, a recent generalisation of the standard setting motivated by the modelling of networked multi-agent systems.

math-ph

Kinetic description and macroscopic limit of swarming dynamics with continuous leader-follower transitions

In this paper, we derive a kinetic description of swarming particle dynamics in an interacting multi-agent system featuring emerging leaders and followers. Agents are classically characterized by their position and velocity plus a continuous parameter quantifying their degree of leadership. The microscopic processes ruling the change of velocity and degree of leadership are independent, non-conservative and non-local in the physical space, so as to account for long-range interactions. Out of the kinetic description, we obtain then a macroscopic model under a hydrodynamic limit reminiscent of that used to tackle the hydrodynamics of weakly dissipative granular gases, thus relying in particular on a regime of small non-conservative and short-range interactions. Numerical simulations in one- and two-dimensional domains show that the limiting macroscopic model is consistent with the original particle dynamics and furthermore can reproduce classical emerging patterns typically observed in swarms.

math-ph

Modelling coevolutionary dynamics in heterogeneous SI epidemiological systems across scales

We develop a new structured compartmental model for the coevolutionary dynamics between susceptible and infectious individuals in heterogeneous SI epidemiological systems. In this model, the susceptible compartment is structured by a continuous variable that represents the level of resistance to infection of susceptible individuals, while the infectious compartment is structured by a continuous variable that represents the viral load of infectious individuals. We first formulate an individual-based model wherein the dynamics of single individuals is described through stochastic processes, which permits a fine-grain representation of individual dynamics and captures stochastic variability in evolutionary trajectories amongst individuals. Next we formally derive the mesoscopic counterpart of this model, which consists of a system of coupled integro-differential equations for the population density functions of susceptible and infectious individuals. Then we consider an appropriately rescaled version of this system and we carry out formal asymptotic analysis to derive the corresponding macroscopic model, which comprises a system of coupled ordinary differential equations for the proportions of susceptible and infectious individuals, the mean level of resistance to infection of susceptible individuals, and the mean viral load of infectious individuals. Overall, this leads to a coherent mathematical representation of the coevolutionary dynamics between susceptible and infectious individuals across scales. We provide well-posedness results for the mesoscopic and macroscopic models, and we show that there is excellent agreement between analytical results on the long-time behaviour of the components of the solution to the macroscopic model, the results of Monte Carlo simulations of the individual-based model, and numerical solutions of the macroscopic model.

q-bio.PE

Network-based kinetic models: Emergence of a statistical description of the graph topology

In this paper, we propose a novel approach that employs kinetic equations to describe the collective dynamics emerging from graph-mediated pairwise interactions in multi-agent systems. We formally show that for large graphs and specific classes of interactions a statistical description of the graph topology, given in terms of the degree distribution embedded in a Boltzmann-type kinetic equation, is sufficient to capture the collective trends of networked interacting systems. This proves the validity of a commonly accepted heuristic assumption in statistically structured graph models, namely that the so-called connectivity of the agents is the only relevant parameter to be retained in a statistical description of the graph topology. Then we validate our results by testing them numerically against real social network data.

physics.soc-ph

Hydrodynamic traffic flow models including random accidents: A kinetic derivation

We present a formal kinetic derivation of a second order macroscopic traffic model from a stochastic particle model. The macroscopic model is given by a system of hyperbolic partial differential equations (PDEs) with a discontinuous flux function, in which the traffic density and the headway are the averaged quantities. A numerical study illustrates the performance of the second order model compared to the particle approach. We also analyse numerically uncertain traffic accidents by considering statistical measures of the solution to the PDEs.

math-ph

Kinetic and macroscopic equations for action potential in neural networks

Starting from the concept of binary interactions between pairs of particles, a kinetic framework for the description of the action potential dynamics on a neural network is proposed. It consists of two coupled levels: the description of a single brain region dynamics and the interactions among different regions. On one side, the pairwise interaction between neurons exchanging membrane potential is statistically described to account for the unmanageable number of neuron synapses within a single brain region. On the other, the network connections accounting for the brain region topology are represented and studied using concepts of the graph theory. Equilibrium and stability of the obtained macroscopic systems are analyzed as well as numerical simulations of the system dynamics are performed in different scenarios. In particular, the latter allows us to observe the influence of the discrete network topology on the membrane potential propagation and synchronization through the different regions, in terms of its spiking characteristics.

physics.bio-ph

An SIR model with viral load-dependent transmission

The viral load is known to be a chief predictor of the risk of transmission of infectious diseases. In this work, we investigate the role of the individuals' viral load in the disease transmission by proposing a new susceptible-infectious-recovered epidemic model for the densities and mean viral loads of each compartment. To this aim, we formally derive the compartmental model from an appropriate microscopic one. Firstly, we consider a multi-agent system in which individuals are identified by the epidemiological compartment to which they belong and by their viral load. Microscopic rules describe both the switch of compartment and the evolution of the viral load. In particular, in the binary interactions between susceptible and infectious individuals, the probability for the susceptible individual to get infected depends on the viral load of the infectious individual. Then, we implement the prescribed microscopic dynamics in appropriate kinetic equations, from which the macroscopic equations for the densities and viral load momentum of the compartments are eventually derived. In the macroscopic model, the rate of disease transmission turns out to be a function of the mean viral load of the infectious population. We analytically and numerically investigate the case that the transmission rate linearly depends on the viral load, which is compared to the classical case of constant transmission rate. A qualitative analysis is performed based on stability and bifurcation theory. Finally, numerical investigations concerning the model reproduction number and the epidemic dynamics are presented.

nlin.AO

Macroscopic limits of non-local kinetic descriptions of vehicular traffic

We study the derivation of macroscopic traffic models out of optimal speed and follow-the-leader particle dynamics as hydrodynamic limits of non-local Povzner-type kinetic equations. As a first step, we show that optimal speed vehicle dynamics produce a first order macroscopic model with non-local flux. Next, we show that non-local follow-the-leader vehicle dynamics have a universal macroscopic counterpart in the second order Aw-Rascle-Zhang traffic model, at least when the non-locality of the interactions is sufficiently small. Finally, we show that the same qualitative result holds also for a general class of follow-the-leader dynamics based on the headway of the vehicles rather than on their speed. We also investigate the correspondence between the solutions to particle models and their macroscopic limits by means of numerical simulations.

cond-mat.stat-mech

Monte Carlo stochastic Galerkin methods for non-Maxwellian kinetic models of multiagent systems with uncertainties

In this paper, we focus on the construction of a hybrid scheme for the approximation of non-Maxwellian kinetic models with uncertainties. In the context of multiagent systems, the introduction of a kernel at the kinetic level is useful to avoid unphysical interactions. The methods here proposed, combine a direct simulation Monte Carlo (DSMC) in the phase space together with stochastic Galerkin (sG) methods in the random space. The developed schemes preserve the main physical properties of the solution together with accuracy in the random space. The consistency of the methods is tested with respect to surrogate Fokker-Planck models that can be obtained in the quasi-invariant regime of parameters. Several applications of the schemes to non-Maxwellian models of multiagent systems are reported.

math.NA

Opinion polarisation in social networks

In this paper, we propose a Boltzmann-type kinetic description of opinion formation on social networks, which takes into account a general connectivity distribution of the individuals. We consider opinion exchange processes inspired by the Sznajd model and related simplifications but we do not assume that individuals interact on a regular lattice. Instead, we describe the structure of the social network statistically, assuming that the number of contacts of a given individual determines the probability that their opinion reaches and influences the opinion of another individual. From the kinetic description of the system, we study the evolution of the mean opinion, whence we find precise analytical conditions under which a \textit{polarisation switch} of the opinions, i.e. a change of sign between the initial and the asymptotic mean opinions, occurs. In particular, we show that a non-zero correlation between the initial opinions and the connectivity of the individuals is necessary to observe polarisation switch. Finally, we validate our analytical results through Monte Carlo simulations of the stochastic opinion exchange processes on the social network.

physics.soc-ph

A statistical mechanics approach to macroscopic limits of car-following traffic dynamics

We study the derivation of macroscopic traffic models from car-following vehicle dynamics by means of hydrodynamic limits of an Enskog-type kinetic description. We consider the superposition of Follow-the-Leader (FTL) interactions and relaxation towards a traffic-dependent Optimal Velocity (OV) and we show that the resulting macroscopic models depend on the relative frequency between these two microscopic processes. If FTL interactions dominate then one gets an inhomogeneous Aw-Rascle-Zhang model, whose (pseudo) pressure and stability of the uniform flow are precisely defined by some features of the microscopic FTL and OV dynamics. Conversely, if the rate of OV relaxation is comparable to that of FTL interactions then one gets a Lighthill-Whitham-Richards model ruled only by the OV function. We further confirm these findings by means of numerical simulations of the particle system and the macroscopic models. Unlike other formally analogous results, our approach builds the macroscopic models as physical limits of particle dynamics rather than assessing the convergence of microscopic to macroscopic solutions under suitable numerical discretisations.

cond-mat.stat-mech

An SIR-like kinetic model tracking individuals' viral load

Mathematical models are formal and simplified representations of the knowledge related to a phenomenon. In classical epidemic models, a neglected aspect is the heterogeneity of disease transmission and progression linked to the viral load of each infectious individual. Here, we attempt to investigate the interplay between the evolution of individuals' viral load and the epidemic dynamics from a theoretical point of view. In the framework of multi-agent systems, we propose a particle stochastic model describing the infection transmission through interactions among agents and the individual physiological course of the disease. Agents have a double microscopic state: a discrete label, that denotes the epidemiological compartment to which they belong and switches in consequence of a Markovian process, and a microscopic trait, representing a normalized measure of their viral load, that changes in consequence of binary interactions or interactions with a background. Specifically, we consider Susceptible--Infected--Removed--like dynamics where infectious individuals may be isolated from the general population and the isolation rate may depend on the viral load sensitivity and frequency of tests. We derive kinetic evolution equations for the distribution functions of the viral load of the individuals in each compartment, whence, via suitable upscaling procedures, we obtain a macroscopic model for the densities and viral load momentum. We perform then a qualitative analysis of the ensuing macroscopic model, and we present numerical tests in the case of both constant and viral load-dependent isolation control. Also, the matching between the aggregate trends obtained from the macroscopic descriptions and the original particle dynamics simulated by a Monte Carlo approach is investigated.

physics.soc-ph