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Andreas Moser

Publications and source records attributed to Andreas Moser.

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Benchmarking Large Language Models for ABAP Code Generation: An Empirical Study on Iterative Improvement by Compiler Feedback

This work investigates the performance of Large Language Models (LLMs) in generating ABAP code. Despite successful applications of generative AI in many programming languages, there are hardly any systematic analyses of ABAP code generation to date. The aim of the study is to empirically analyze to what extent various LLMs can generate syntactically correct and functional ABAP code, how effectively they use compiler feedback for iterative improvement, and which task types pose special challenges. For this purpose, a benchmark with 180 tasks is conducted, consisting of adapted HumanEval tasks and practical SAP scenarios. The results show significant performance differences between the models: more powerful LLMs achieve success rates of around 75% after several iterations and benefit greatly from compiler feedback, while smaller models perform significantly weaker. Overall, the study highlights the high potential of powerful LLMs for ABAP development processes, especially in iterative error correction.

cs.SE

Modeling effect of GABAergic current in a basal ganglia computational model

Electrical high frequency stimulation (HFS) of deep brain regions is a method shown to be clinically effective in different types of movement and neurological disorders. In order to shed light on its mode of action a computational model of the basal ganglia network coupled the HFS as injection current into the cells of the subthalamic nucleus (STN). Its overall increased activity rendered a faithful transmission of sensorimotor input through thalamo-cortical relay cells possible. Our contribution uses this model by Rubin and Terman (J Comput Neurosci, 16, 211-223, 2004) as a starting point and integrates recent findings on the importance of the extracellular concentrations of the inhibiting neurotransmitter GABA. We are able to show in this computational study that besides electrical stimulation a high concentration of GABA and its resulting conductivity in STN cells is able to re-establish faithful thalamocortical relaying, which otherwise broke down in the simulated parkinsonian state.

q-bio.NC