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Andrew L. Krause

Publications and source records attributed to Andrew L. Krause.

26 records · Page 2Linked to original sources

Unstaggered-staggered solitons on one- and two-dimensional two-component discrete nonlinear Schrödinger lattices

We study coupled unstaggered-staggered soliton pairs emergent from a system of two coupled discrete nonlinear Schrödinger (DNLS) equations with the self-attractive on-site self-phase-modulation nonlinearity, coupled by the repulsive cross-phase-modulation interaction, on 1D and 2D lattice domains. These mixed modes are of a "symbiotic" type, as each component in isolation may only carry ordinary unstaggered solitons. While most work on DNLS systems addressed symmetric on-site-centered fundamental solitons, these models give rise to a variety of other excited states, which may also be stable. The simplest among them are antisymmetric states in the form of discrete twisted solitons, which have no counterparts in the continuum limit. In the extension to 2D lattice domains, a natural counterpart of the twisted states are vortical solitons. We first introduce a variational approximation (VA) for the solitons, and then correct it numerically to construct exact stationary solutions, which are then used as initial conditions for simulations to check if the stationary states persist under time evolution. Two-component solutions obtained include (i) 1D fundamental-twisted and twisted-twisted soliton pairs, (ii) 2D fundamental-fundamental soliton pairs, and (iii) 2D vortical-vortical soliton pairs. We also highlight a variety of other transient dynamical regimes, such as breathers and amplitude death. The findings apply to modeling binary Bose-Einstein condensates, loaded in a deep lattice potential, with identical or different atomic masses of the two components, and arrays of bimodal optical waveguides.

nlin.PS↗

From One Pattern into Another: Analysis of Turing Patterns in Heterogeneous Domains via WKBJ

Pattern formation from homogeneity is well-studied, but less is known concerning symmetry-breaking instabilities in heterogeneous media. It is nontrivial to separate observed spatial patterning due to inherent spatial heterogeneity from emergent patterning due to nonlinear instability. We employ WKBJ asymptotics to investigate Turing instabilities for a spatially heterogeneous reaction-diffusion system, and derive conditions for instability which are local versions of the classical Turing conditions We find that the structure of unstable modes differs substantially from the typical trigonometric functions seen in the spatially homogeneous setting. Modes of different growth rates are localized to different spatial regions. This localization helps explain common amplitude modulations observed in simulations of Turing systems in heterogeneous settings. We numerically demonstrate this theory, giving an illustrative example of the emergent instabilities and the striking complexity arising from spatially heterogeneous reaction-diffusion systems. Our results give insight both into systems driven by exogenous heterogeneity, as well as successive pattern forming processes, noting that most scenarios in biology do not involve symmetry breaking from homogeneity, but instead consist of sequential evolutions of heterogeneous states. The instability mechanism reported here precisely captures such evolution, and extends Turing's original thesis to a far wider and more realistic class of systems.

nlin.PS↗

Turing conditions for pattern forming systems on evolving manifolds

The study of pattern-forming instabilities in reaction-diffusion systems on growing or otherwise time-dependent domains arises in a variety of settings, including applications in developmental biology, spatial ecology, and experimental chemistry. Analyzing such instabilities is complicated, as there is a strong dependence of any spatially homogeneous base states on time, and the resulting structure of the linearized perturbations used to determine the onset of instability is inherently non-autonomous. We obtain general conditions for the onset and structure of diffusion driven instabilities in reaction-diffusion systems on domains which evolve in time, in terms of the time-evolution of the Laplace-Beltrami spectrum for the domain and functions which specify the domain evolution. Our results give sufficient conditions for diffusive instabilities phrased in terms of differential inequalities which are both versatile and straightforward to implement, despite the generality of the studied problem. These conditions generalize a large number of results known in the literature, such as the algebraic inequalities commonly used as a sufficient criterion for the Turing instability on static domains, and approximate asymptotic results valid for specific types of growth, or specific domains. We demonstrate our general Turing conditions on a variety of domains with different evolution laws, and in particular show how insight can be gained even when the domain changes rapidly in time, or when the homogeneous state is oscillatory, such as in the case of Turing-Hopf instabilities. Extensions to higher-order spatial systems are also included as a way of demonstrating the generality of the approach.

nlin.PS↗

Amplitude death criteria for coupled complex Ginzburg-Landau systems

Amplitude death, which occurs in a system when one or more macroscopic wavefunctions collapse to zero, has been observed in mutually coupled solid-state lasers, analog circuits, and thermoacoustic oscillators, to name a few applications. While studies have considered amplitude death on oscillator systems and in externally forced complex Ginzburg-Landau systems, a route to amplitude death has not been studied in autonomous continuum systems. We derive simple analytic conditions for the onset of amplitude death of one macroscopic wavefunction in a system of two coupled complex Ginzburg-Landau equations with general nonlinear self- and cross-interaction terms. Our results give a more general theoretical underpinning for recent amplitude death results reported in the literature, and suggest an approach for tuning parameters in such systems so that they either permit or prohibit amplitude death of a wavefunction (depending on the application). Numerical simulation of the coupled complex Ginzburg-Landau equations, for examples including cubic, cubic-quintic, and saturable nonlinearities, is used to illustrate the analytical results.

nlin.PS↗

Coupled complex Ginzburg-Landau systems with saturable nonlinearity and asymmetric cross-phase modulation

We formulate and study dynamics from a complex Ginzburg-Landau system with saturable nonlinearity, including asymmetric cross-phase modulation (XPM) parameters. Such equations can model phenomena described by complex Ginzburg-Landau systems under the added assumption of saturable media. When the saturation parameter is set to zero, we recover a general complex cubic Ginzburg-Landau system with XPM. We first derive conditions for the existence of bounded dynamics, approximating the absorbing set for solutions. We use this to then determine conditions for amplitude death of a single wavefunction. We also construct exact plane wave solutions, and determine conditions for their modulational instability. In a degenerate limit where dispersion and nonlinearity balance, we reduce our system to a saturable nonlinear Schrödinger system with XPM parameters, and we demonstrate the existence and behavior of spatially heterogeneous stationary solutions in this limit. Using numerical simulations we verify the aforementioned analytical results, while also demonstrating other interesting emergent features of the dynamics, such as spatiotemporal chaos in the presence of modulational instability. In other regimes, coherent patterns including uniform states or banded structures arise, corresponding to certain stable stationary states. For sufficiently large yet equal XPM parameters, we observe a segregation of wavefunctions into different regions of the spatial domain, while when XPM parameters are large and take different values, one wavefunction may decay to zero in finite time over the spatial domain (in agreement with the amplitude death predicted analytically). While saturation will often regularize the dynamics, such transient dynamics can still be observed - and in some cases even prolonged - as the saturability of the media is increased, as the saturation may act to slow the timescale.

nlin.PS↗

Bifurcations and dynamics emergent from lattice and continuum models of bioactive porous media

We study dynamics emergent from a two-dimensional reaction--diffusion process modelled via a finite lattice dynamical system, as well as an analogous PDE system, involving spatially nonlocal interactions. These models govern the evolution of cells in a bioactive porous medium, with evolution of the local cell density depending on a coupled quasi--static fluid flow problem. We demonstrate differences emergent from the choice of a discrete lattice or a continuum for the spatial domain of such a process. We find long--time oscillations and steady states in cell density in both lattice and continuum models, but that the continuum model only exhibits solutions with vertical symmetry, independent of initial data, whereas the finite lattice admits asymmetric oscillations and steady states arising from symmetry-breaking bifurcations. We conjecture that it is the structure of the finite lattice which allows for more complicated asymmetric dynamics. Our analysis suggests that the origin of both types of oscillations is a nonlocal reaction-diffusion mechanism mediated by quasi-static fluid flow.

q-bio.TO↗

Chaotic dynamics in the planar gravitational many-body problem with rigid body rotations

The discovery of Pluto's small moons in the last decade brought attention to the dynamics of the dwarf planet's satellites. With such systems in mind, we study a planar $N$-body system in which all the bodies are point masses, except for a single rigid body. We then present a reduced model consisting of a planar $N$-body problem with the rigid body treated as a 1D continuum (i.e. the body is treated as a rod with an arbitrary mass distribution). Such a model provides a good approximation to highly asymmetric geometries, such as the recently observed interstellar asteroid 'Oumuamua, but is also amenable to analysis. We analytically demonstrate the existence of homoclinic chaos in the case where one of the orbits is nearly circular by way of the Melnikov method, and give numerical evidence for chaos when the orbits are more complicated. We show that the extent of chaos in parameter space is strongly tied to the deviations from a purely circular orbit. These results suggest that chaos is ubiquitous in many-body problems when one or more of the rigid bodies exhibits non-spherical and highly asymmetric geometries. The excitation of chaotic rotations does not appear to require tidal dissipation, obliquity variation, or orbital resonance. Such dynamics give a possible explanation for routes to chaotic dynamics observed in $N$-body systems such as the Pluto system where some of the bodies are highly non-spherical.

astro-ph.EP↗

Lattice and Continuum Modelling of a Bioactive Porous Tissue Scaffold

A contemporary procedure to grow artificial tissue is to seed cells onto a porous biomaterial scaffold and culture it within a perfusion bioreactor to facilitate the transport of nutrients to growing cells. Typical models of cell growth for tissue engineering applications make use of spatially homogeneous or spatially continuous equations to model cell growth, flow of culture medium, nutrient transport, and their interactions. The network structure of the physical porous scaffold is often incorporated through parameters in these models, either phenomenologically or through techniques like mathematical homogenization. We derive a model on a square grid lattice to demonstrate the importance of explicitly modelling the network structure of the porous scaffold, and compare results from this model with those from a modified continuum model from the literature. We capture two-way coupling between cell growth and fluid flow by allowing cells to block pores, and by allowing the shear stress of the fluid to affect cell growth and death. We explore a range of parameters for both models, and demonstrate quantitative and qualitative differences between predictions from each of these approaches, including spatial pattern formation and local oscillations in cell density present only in the lattice model. These differences suggest that for some parameter regimes, corresponding to specific cell types and scaffold geometries, the lattice model gives qualitatively different model predictions than typical continuum models. Our results inform model selection for bioactive porous tissue scaffolds, aiding in the development of successful tissue engineering experiments and eventually clinically successful technologies.

q-bio.TO↗