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David Juher

Publications and source records attributed to David Juher.

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Finite capture and the closure of roots of restricted polynomials

We study how a countable algebraic root set passes to a fractal connectedness locus. Let $D_n=\{-n+1,-n+2,\ldots,n-1\}$, and let $R_n$ be the set of roots of monic polynomials whose non-leading coefficients lie in $D_n$. We study $\overline{R_n}\setminus\overline{\mathbb{D}}$. Outside the closed unit disk this set equals a connectedness locus $M_n$ for a collinear affine iterated function system, or equivalently the zero set of reciprocal power series $1+\sum_{k\ge1} d_k c^{-k}$ with $d_k\in D_n$. For non-real parameters in the lens $X_n=\{\,c\in\mathbb{C}\setminus\overline{\mathbb{D}}:\ |c\pm1|<\sqrt{2n}\,\}$ we construct a canonical trap and enclosure for the associated difference attractor and use them to define finite-capture sets $\Theta_k(n)$ for the marked point $2c$. Our main result is the uniform inclusion $\overline{\Theta_k(n)}\cap(X_n\setminus\mathbb{R})\subset\Theta_{k+2}(n)$ for every $k\ge0$. Consequently, $(M_n\cap X_n)\setminus\mathbb{R}$ is exactly the closure of the finite-capture locus. The paper combines explicit trap geometry with certified inverse search. Moreover, $M_n\setminus\mathbb{R}\subset X_n$ for every $n\ge20$, and this is sharp for $2\le n\le19$. Thus, for $n\ge20$, the non-real part of $\overline{R_n}\setminus\overline{\mathbb{D}}$ is exactly the closure of the finite-capture locus.

math.DS

Collinear Fractals and Bandt's Conjecture

For a complex parameter $c$ outside the unit disk and an integer $n\ge2$, we examine the $n$-ary collinear fractal $E(c,n)$, defined as the attractor of the iterated function system $\{\mbox{$f_k \colon \mathbb{C} \longrightarrow \mathbb{C}$}\}_{k=1}^n$, where $f_k(z):=1+n-2k+c^{-1}z$. We investigate some topological features of the connectedness locus $\mathcal{M}_n$, similar to the Mandelbrot set, defined as the set of those $c$ for which $E(c,n)$ is connected. In particular, we provide a detailed answer to an open question posed by Calegari, Koch, and Walker in 2017. We also extend and refine the technique of the covering property by Solomyak and Xu to any $n\ge2$. We use it to show that a nontrivial portion of $\mathcal{M}_n$ is regular-closed. When $n\ge21$, we enhance this result by showing that, in fact, the whole $\mathcal{M}_n\setminus\mathbb{R}$ lies within the closure of its interior, thus proving that the generalized Bandt's conjecture is true.

math.DS

Characterization of the tree cycles with minimum positive entropy for any period

Consider, for any integer $n\ge3$, the set $\text{Pos}_n$ of all $n$-periodic tree patterns with positive topological entropy and the set $\text{Irr}_n\subset\text{Pos}_n$ of all $n$-periodic irreducible tree patterns. The aim of this paper is to determine the elements of minimum entropy in the families $\text{Pos}_n$, $\text{Irr}_n$ and $\text{Pos}_n\setminus\text{Irr}_n$. Let $\lambda_n$ be the unique real root of the polynomial $x^n-2x-1$ in $(1,+\infty)$. We explicitly construct an irreducible $n$-periodic tree pattern $\mathcal{Q}_n$ whose entropy is $\log(\lambda_n)$. We prove that this entropy is minimum in $\text{Pos}_n$. Since the pattern $\mathcal{Q}_n$ is irreducible, $\mathcal{Q}_n$ also minimizes the entropy in the family $\text{Irr}_n$. We also prove that the minimum positive entropy in the set $\text{Pos}_n\setminus\text{Irr}_n$ (which is nonempty only for composite integers $n\ge6$) is $\log(\lambda_{n/p})/p$, where $p$ is the least prime factor of $n$.

math.DS

Saddle-node bifurcation of limit cycles in an epidemic model with two levels of awareness

In this paper we study the appearance of bifurcations of limit cycles in an epidemic model with two types of aware individuals. All the transition rates are constant except for the alerting decay rate of the most aware individuals and the rate of creation of the less aware individuals, which depend on the disease prevalence in a non-linear way. For the ODE model, the numerical computation of the limit cycles and the study of their stability are made by means of the Poincar\'e map. Moreover, sufficient conditions for the existence of an endemic equilibrium are also obtained. These conditions involve a rather natural relationship between the transmissibility of the disease and that of awareness. Finally, stochastic simulations of the model under a very low rate of imported cases are used to confirm the scenarios of bistability (endemic equilibrium and limit cycle) observed in the solutions of the ODE model.

q-bio.PE

Robustness of behaviourally-induced oscillations in epidemic models under a low rate of imported cases

This paper is concerned with the robustness of the sustained oscillations predicted by an epidemic ODE model defined on contact networks. The model incorporates the spread of awareness among individuals and, moreover, a small inflow of imported cases. These cases prevent stochastic extinctions when we simulate the epidemics and, hence, they allow to check whether the average dynamics for the fraction of infected individuals are accurately predicted by the ODE model. Stochastic simulations confirm the existence of sustained oscillations for different types of random networks, with a sharp transition from a non-oscillatory asymptotic regime to a periodic one as the alerting rate of susceptible individuals increases from very small values. This abrupt transition to periodic epidemics of high amplitude is quite accurately predicted by the Hopf-bifurcation curve computed from the ODE model using the alerting rate and the infection transmission rate for aware individuals as tuning parameters.

q-bio.PE

SIS Epidemics in Multilayer-based Temporal Networks

To improve the accuracy of network-based SIS models we introduce and study a multilayer representation of a time-dependent network. In particular, we assume that individuals have their long-term (permanent) contacts that are always present, identifying in this way the first network layer. A second network layer also exists, where the same set of nodes can be connected by occasional links, created with a given probability. While links of the first layer are permanent, a link of the second layer is only activated with some probability and under the condition that the two nodes, connected by this link, are simultaneously participating to the temporary link. We develop a model for the SIS epidemic on this time-dependent network, analyze equilibrium and stability of the corresponding mean-field equations, and shed some light on the role of the temporal layer on the spreading process.

physics.soc-ph

A network epidemic model with preventive rewiring: comparative analysis of the initial phase

This paper is concerned with stochastic SIR and SEIR epidemic models on random networks in which individuals may rewire away from infected neighbors at some rate $ω$ (and reconnect to non-infectious individuals with probability $α$ or else simply drop the edge if $α=0$), so-called preventive rewiring. The models are denoted SIR-$ω$ and SEIR-$ω$, and we focus attention on the early stages of an outbreak, where we derive expression for the basic reproduction number $R_0$ and the expected degree of the infectious nodes $E(D_I)$ using two different approximation approaches. The first approach approximates the early spread of an epidemic by a branching process, whereas the second one uses pair approximation. The expressions are compared with the corresponding empirical means obtained from stochastic simulations of SIR-$ω$ and SEIR-$ω$ epidemics on Poisson and scale-free networks. Without rewiring of exposed nodes, the two approaches predict the same epidemic threshold and the same $E(D_I)$ for both types of epidemics, the latter being very close to the mean degree obtained from simulated epidemics over Poisson networks. Above the epidemic threshold, pairwise models overestimate the value of $R_0$ computed from simulations, which turns out to be very close to the one predicted by the branching process approximation. When exposed individuals also rewire with $α> 0$ (perhaps unaware of being infected), the two approaches give different epidemic thresholds, with the branching process approximation being more in agreement with simulations.

q-bio.PE

Creating and controlling overlap in two-layer networks. Application to a mean-field SIS epidemic model with awareness dissemination

We study the properties of the potential overlap between two networks $A,B$ sharing the same set of $N$ nodes (a two-layer network) whose respective degree distributions $p_A(k), p_B(k)$ are given. Defining the overlap coefficient $\alpha$ as the Jaccard index, we derive upper bounds for the minimum and maximum overlap coefficient in terms of $p_A(k)$, $p_B(k)$ and $N$. We also present an algorithm based on cross-rewiring of links to obtain a two-layer network with any prescribed $\alpha$ inside the permitted range. Finally, to illustrate the importance of the overlap for the dynamics of interacting contagious processes, we derive a mean-field model for the spread of an SIS epidemic with awareness against infection over a two-layer network, containing $\alpha$ as a parameter. A simple analytical relationship between $\alpha$ and the basic reproduction number follows. Stochastic simulations are presented to assess the accuracy of the upper bounds of $\alpha$ and the predictions of the mean-field epidemic model.

physics.soc-ph

Analysis of an epidemic model with awareness decay on regular random networks

The existence of a die-out threshold (different from the classic disease-invasion one) defining a region of slow extinction of an epidemic has been proved elsewhere for susceptible-aware-infectious-susceptible models without awareness decay, through bifurcation analysis. By means of an equivalent mean-field model defined on regular random networks, we interpret the dynamics of the system in this region and prove that the existence of bifurcation for this second epidemic threshold crucially depends on the absence of awareness decay. We show that the continuum of equilibria that characterizes the slow die-out dynamics collapses into a unique equilibrium when a constant rate of awareness decay is assumed, no matter how small, and that the resulting bifurcation from the disease-free equilibrium is equivalent to that of standard epidemic models. We illustrate these findings with continuous-time stochastic simulations on regular random networks with different degrees. Finally, the behaviour of solutions with and without decay in awareness is compared around the second epidemic threshold for a small rate of awareness decay.

q-bio.QM

Volume entropy for minimal presentations of surface groups in all ranks

We study the volume entropy of a class of presentations (including the classical ones) for all surface groups, called \emph{minimal geometric presentations}. We rediscover a formula first obtained by Cannon and Wagreich with the computation in a non published manuscript by Cannon. The result is surprising: an explicit polynomial of degree $n$, the rank of the group, encodes the volume entropy of all classical presentations of surface groups. The approach we use is completely different. It is based on a dynamical system construction following an idea due to Bowen and Series and extended to all geometric presentations in. The result is an explicit formula for the volume entropy of minimal presentations for all surface groups, showing a polynomial dependence in the rank $n>2$. We prove that for a surface group $G_n$ of rank $n$ with a classical presentation $P_n$ the volume entropy is $\log(λ_n)$, where $λ_n$ is the unique real root larger than one of the polynomial \[ x^{n} - 2(n - 1) \sum_{j=1}^{n-1} x^{j} + 1. \]

math.DS