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G. Krings

Publications and source records attributed to G. Krings.

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Significant Scales in Community Structure

Many complex networks show signs of modular structure, uncovered by community detection. Although many methods succeed in revealing various partitions, it remains difficult to detect at what scale some partition is significant. This problem shows foremost in multi-resolution methods. We here introduce an efficient method for scanning for resolutions in one such method. Additionally, we introduce the notion of "significance" of a partition, based on subgraph probabilities. Significance is independent of the exact method used, so could also be applied in other methods, and can be interpreted as the gain in encoding a graph by making use of a partition. Using significance, we can determine "good" resolution parameters, which we demonstrate on benchmark networks. Moreover, optimizing significance itself also shows excellent performance. We demonstrate our method on voting data from the European Parliament. Our analysis suggests the European Parliament has become increasingly ideologically divided and that nationality plays no role.

physics.soc-ph

Urban Gravity: a Model for Intercity Telecommunication Flows

We analyze the anonymous communication patterns of 2.5 million customers of a Belgian mobile phone operator. Grouping customers by billing address, we build a social network of cities, that consists of communications between 571 cities in Belgium. We show that inter-city communication intensity is characterized by a gravity model: the communication intensity between two cities is proportional to the product of their sizes divided by the square of their distance.

physics.soc-ph

The Role of Second Trials in Cascades of Information over Networks

We study the propagation of information in social networks. To do so, we focus on a cascade model where nodes are infected with {probability $p_1$ after their first contact with the information and with probability $p_2$ at all subsequent contacts.} The diffusion starts from one random node and leads to a cascade of infection. It is shown that first and {subsequent} trials play different roles in the propagation and that the size of the cascade depends in a non-trivial way on $p_1$, $p_2$ and on the network structure. Second trials are shown to amplify the propagation in dense parts of the network while first trials are {dominant for the exploration of} new parts of the network and launching new seeds of infection.

physics.soc-ph