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G. Timár

Publications and source records attributed to G. Timár.

6 recordsLinked to original sources

Localization of nonbacktracking centrality on dense subgraphs of sparse networks

The nonbacktracking matrix, and the related nonbacktracking centrality (NBC) play a crucial role in models of percolation-type processes on networks, such as non-recurrent epidemics. Here we study the localization of NBC in infinite sparse networks that contain an arbitrary finite subgraph. Assuming the local tree-likeness of the enclosing network, and that branches emanating from the finite subgraph do not intersect at finite distances, we show that the largest eigenvalue of the nonbacktracking matrix of the composite network is equal to the highest of the two largest eigenvalues: that of the finite subgraph and of the enclosing network. In the localized state, when the largest eigenvalue of the subgraph is the highest of the two, we derive explicit expressions for the NBCs of nodes in the subgraph and other nodes in the network. In this state, nonbacktracking centrality is concentrated on the subgraph and its immediate neighbourhood in the enclosing network. We obtain simple, exact formulas in the case where the enclosing network is uncorrelated. We find that the mean NBC decays exponentially around the finite subgraph, at a rate which is independent of the structure of the enclosing network, contrary to what was found for the localization of the principal eigenvector of the adjacency matrix. Numerical simulations confirm that our results provide good approximations even in moderately sized, loopy, real-world networks.

physics.soc-ph

Approximating nonbacktracking centrality and localization phenomena in large networks

Message-passing theories have proved to be invaluable tools in studying percolation, non-recurrent epidemics and similar dynamical processes on real-world networks. At the heart of the message-passing method is the nonbacktracking matrix whose largest eigenvalue, the corresponding eigenvector, and the closely related nonbacktracking centrality play a central role in determining how the given dynamical model behaves. Here we propose a degree-class-based method to approximate these quantities using a smaller matrix related to the joint degree-degree distribution of neighbouring nodes. Our findings suggest that in most networks degree-degree correlations beyond nearest neighbour are actually not strong, and our first-order description already results in accurate estimates, particularly when message-passing itself is a good approximation to the original model in question, that is when the number of short cycles in the network is sufficiently low. We show that localization of the nonbacktracking centrality is also captured well by our scheme, particularly in large networks. Our method provides an alternative to working with the full nonbacktracking matrix in very large networks where this may not be possible due to memory limitations.

physics.soc-ph

Degree dependent transmission rates in epidemic processes

The outcome of SIR epidemics with heterogeneous infective lifetimes, or heterogeneous susceptibilities, can be mapped onto a directed percolation process on the underlying contact network. In this paper we study SIR models where heterogeneity is a result of the degree dependence of disease transmission rates. We develop numerical methods to determine the epidemic threshold, the epidemic probability and epidemic size close to the threshold for configuration model contact networks with arbitrary degree distribution and an arbitrary matrix of transmission rates (dependent on transmitting and receiving node degree). For the special case of separable transmission rates we obtain analytical expressions for these quantities. We propose a categorization of spreading processes based on the ratio of the probability of an epidemic and the expected size of an epidemic, and demonstrate that this ratio has a complex dependence on the degree distribution and the degree-dependent transmission rates. For scale-free contact networks and transmission rates that are power functions of transmitting and receiving node degrees, the epidemic threshold may be finite even when the degree distribution powerlaw exponent is below $γ< 3$. We give an expression, in terms of the degree distribution and transmission rate exponents, for the limit at which the epidemic threshold vanishes. We find that the expected epidemic size and the probability of an epidemic may grow nonlinearly above the epidemic threshold, with exponents that depend not only on the degree distribution powerlaw exponent, but on the parameters of the transmission rate degree dependence functions, in contrast to ordinary directed percolation and previously studied variations of the SIR model.

physics.soc-ph

Targeted Damage to Interdependent Networks

The giant mutually connected component (GMCC) of an interdependent or multiplex network collapses with a discontinuous hybrid transition under random damage to the network. If the nodes to be damaged are selected in a targeted way, the collapse of the GMCC may occur significantly sooner. Finding the minimal damage set which destroys the largest mutually connected component of a given interdependent network is a computationally prohibitive simultaneous optimization problem. We introduce a simple heuristic strategy -- Effective Multiplex Degree -- for targeted attack on interdependent networks that leverages the indirect damage inherent in multiplex networks to achieve a damage set smaller than that found by any other non computationally intensive algorithm. We show that the intuition from single layer networks that decycling (damage of the $2$-core) is the most effective way to destroy the giant component, does not carry over to interdependent networks, and in fact such approaches are worse than simply removing the highest degree nodes.

physics.soc-ph

Sensitivity of directed networks to the addition and pruning of edges and vertices

We study the sensitivity of directed complex networks to the addition and pruning of edges and vertices and introduce the susceptibility, which quantifies this sensitivity. We show that topologically different parts of a directed network have different sensitivity to the addition and pruning of edges and vertices and, therefore, they are characterized by different susceptibilities. These susceptibilities diverge at the critical point of the directed percolation transition, signaling the appearance (or disappearance) of the giant strongly connected component in the infinite size limit. We demonstrate this behavior in randomly damaged real and synthetic directed complex networks, such as the World Wide Web, Twitter, the \emph{Caenorhabditis elegans} neural network, directed Erdős-Rényi graphs, and others. We reveal a non-monotonous dependence of the sensitivity to random pruning of edges or vertices in the case of \emph{Caenorhabditis elegans} and Twitter that manifests specific structural peculiarities of these networks. We propose the measurements of the susceptibilities during the addition or pruning of edges and vertices as a new method for studying structural peculiarities of directed networks.

cond-mat.dis-nn

Mapping the Structure of Directed Networks: Beyond the "Bow-tie" Diagram

We reveal a hierarchical, multilayer organization of finite components -- i.e., tendrils and tubes -- around the giant connected components in directed networks and propose efficient algorithms allowing one to uncover the entire organization of key real-world directed networks, such as the World Wide Web, the neural network of \emph{Caenorhabditis elegans}, and others. With increasing damage, the giant components decrease in size while the number and size of tendril layers increase, enhancing the susceptibility of the networks to damage.

cond-mat.dis-nn