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Iulia Martina Bulai

Publications and source records attributed to Iulia Martina Bulai.

6 recordsLinked to original sources

Dynamical Sampling in Graph Paley--Wiener Spaces

We study dynamical sampling for graph signals using the spectral theory of the normalized graph Laplacian. Graph Paley-Wiener spaces GPW_w are defined as spectral subspaces associated with a bandwidth parameter w, and reconstruction is studied from measurements generated by iterates of bounded operators leaving these spaces invariant. For the lattice graph Z, we give an explicit spectral decomposition of the normalized Laplacian, identify the corresponding Graph Paley--Wiener spaces with classical bandlimited spaces on the torus, and recover the classical sampling theorem in this setting. Using frames of operator orbits, we construct dynamical frames for GPW_w(Z) and derive reconstruction formulas. We also discuss the extension to Z^n, where the bandwidth region is described by a sublevel set of the Laplacian symbol. Finally, for finite graphs, we formulate dynamical recovery in GPW_w(G)\) in terms of finite frames generated by iterates of the graph operator. Using the path graph P_N as a numerical model, we study how the number of iterates and the spatial distribution of the sampled vertices affect the optimal frame bounds and the conditioning of the resulting dynamical frame.

math.CA↗

Optimization of the total tumor population under Gompertz growth

We study optimal control problems for a stationary reaction--diffusion model describing the spatial distribution of a tumor cell population with Gompertz growth. The control $m(x)$ represents a treatment term acting as a density-dependent removal rate and it is subject to $L^{1}-L^{\infty}$ constraints. When the intrinsic growth rate is constant, the uniform distribution of the treatment is shown to be the unique minimizer. For the maximization problem, we prove that every optimal control is of bang-bang type. In addition, we show that in the one dimensional case and for sufficiently large diffusion rates, the positivity set of optimal controls is an interval sticking to one of the extrema of the domain. Finally, numerical simulations complement the theoretical analysis and explore regimes that are not fully covered by the results proved in the paper. The computations confirm the bang-bang structure of maximizers, and illustrate how the shape of optimal controls and the associated states are affected by spatial heterogeneity in the growth rate, localized admissible treatment regions, and the diffusion coefficient. Moreover, they reveal a monotone dependence of the optimized total population on the diffusion coefficient: this is a new phenomenon with respect to the logistic setting.

math.AP↗

Beyond Single-Window Graph Fourier Analysis

We introduce a multi-windowed graph Fourier transform (MWGFT) for the joint vertex-frequency analysis of signals defined on graphs. Building on generalized translation and modulation induced by the graph Laplacian, the proposed framework extends the windowed graph Fourier transform by allowing multiple analysis and synthesis windows. Exact reconstruction formulas are derived for complex-valued graph signals, together with sufficient and computable conditions guaranteeing stable invertibility. The associated families of windowed graph Fourier atoms are shown to form frames for the space of graph signals. Numerical experiments on synthetic and real world graphs confirm exact reconstruction up to machine precision and demonstrate improved stability and vertex-frequency localization compared to single-window constructions, particularly on irregular graph topologies.

math.CA↗

A geometric analysis of the SIRS compartmental model with fast information and misinformation spreading

We propose a novel slow-fast SIRS compartmental model with demography, by coupling a slow disease spreading model and a fast information and misinformation spreading model. Beside the classes of susceptible, infected and recovered individuals of a common SIRS model, here we define three new classes related to the information spreading model, e.g. unaware individuals, misinformed individuals and individuals who are skeptical to disease-related misinformation. Under our assumptions, the system evolves on two time scales. We completely characterize its asymptotic behaviour with techniques of Geometric Singular Perturbation Theory (GSPT). We exploit the time scale separation to analyse two lower dimensional subsystem separately. First, we focus on the analysis of the fast dynamics and we find three equilibrium point which are feasible and stable under specific conditions. We perform a theoretical bifurcation analysis of the fast system to understand the relations between these three equilibria when varying specific parameters of the fast system. Secondly, we focus on the evolution of the slow variables and we identify three branches of the critical manifold, which are described by the three equilibria of the fast system. We fully characterize the slow dynamics on each branch. Moreover, we show how the inclusion of (mis)information spread may negatively or positively affect the evolution of the epidemic, depending on whether the slow dynamics evolves on the second branch of the critical manifold, related to the skeptical-free equilibrium or on the third one, related to misinformed-free equilibrium, respectively. We conclude with numerical simulations which showcase our analytical results.

math.DS↗

Hierarchical bases preconditioner to enhance convergence of the CFIE with multiscale meshes

A hierarchical quasi-Helmholtz decomposition, originally developed to address the low-frequency and dense-discretization breakdowns for the EFIE, is applied together with an algebraic preconditioner to improve the convergence of the CFIE in multiscale problems. The effectiveness of the proposed method is studied first on some simple examples; next, test on real-life cases up to several hundreds wavelengths show its good performance.

math.NA↗

Comparing disease control policies for interacting wild populations

We consider interacting population systems of predator-prey type, presenting four models of control strategies for epidemics among the prey. In particular to contain the transmissible disease, safety niches are considered, assuming they lessen the disease spread, but do not protect prey from predators. This represents a novelty with respect to standard ecosystems where the refuge prevents predators' attacks. The niche is assumed either to protect the healthy individuals, or to hinder the infected ones to get in contact with the susceptibles, or finally to reduce altogether contacts that might lead to new cases of the infection. In addition a standard culling procedure is also analysed. The effectiveness of the different strategies are compared. Probably the environments providing a place where disease carriers cannot come in contact with the healthy individuals, or where their contact rates are lowered, seem to preferable for disease containment.

q-bio.PE↗