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Jiwei Fu

Publications and source records attributed to Jiwei Fu.

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SETA: Scaling Environments for Terminal Agents

Large language models (LLMs) are rapidly shifting toward agents that solve tasks through diverse interfaces, including web and graphical user interfaces (GUIs). Among these, the terminal command line provides a text-based, general-purpose interface, covering tasks from system operations to data science and machine learning. However, scaling terminal-agent training remains challenging, as it requires diverse and coherent task instructions, executable environments, and reliable verification, while lacking naturally grounded supervision data. In this work, we propose SETA, a scalable framework for generating verifiable terminal environments for reinforcement learning (RL). The framework consists of two pipelines sharing a unified verification mechanism: SETA-Synth converts diverse sources into standardized RL environments, and SETA-Evol further expands from existing environments with adaptive control of difficulty and diversity. Together, we construct and release SETA-Env, the largest open-source verifiable terminal RL dataset to date, containing over 4,500 environments. We evaluate our dataset by training Qwen3-8B with GRPO on SETA-Env, achieving 12% pass rate on Terminal-Bench 2.0, the best reported result for an RL-trained model at the 8B scale. We further observe gains on DeepSeek-V4-Flash under the same terminal agent harness, with pass@1 on Terminal-Bench 2.0 improving from 40% to 43% and pass@5 improving from 54% to 58%. These results demonstrate that SETA- Env provides high-quality training environments for terminal agents and serves as a valuable resource for advancing research on terminal-based agent learning.

cs.AI

Genotype-Phenotype Integration through Machine Learning and Personalized Gene Regulatory Networks for Cancer Metastasis Prediction

Metastasis is the leading cause of cancer-related mortality, yet most predictive models rely on shallow architectures and neglect patient-specific regulatory mechanisms. Here, we integrate classical machine learning and deep learning to predict metastatic potential across multiple cancer types. Gene expression profiles from the Cancer Cell Line Encyclopedia were combined with a transcription factor-target prior from DoRothEA, focusing on nine metastasis-associated regulators. After selecting differential genes using the Kruskal-Wallis test, ElasticNet, Random Forest, and XGBoost models were trained for benchmarking. Personalized gene regulatory networks were then constructed using PANDA and LIONESS and analyzed through a graph attention neural network (GATv2) to learn topological and expression-based representations. While XGBoost achieved the highest AUROC (0.7051), the GNN captured non-linear regulatory dependencies at the patient level. These results demonstrate that combining traditional machine learning with graph-based deep learning enables a scalable and interpretable framework for metastasis risk prediction in precision oncology.

q-bio.OT