Searcharxiv⌕ Search

arXiv subjects

Lidia A. Braunstein

Publications and source records attributed to Lidia A. Braunstein.

At least 19 recordsLinked to original sources

Critical behavior of cascading failures in overloaded networks

While network abrupt breakdowns due to overloads and cascading failures have been studied extensively, the critical exponents and the universality class of such phase transitions have not been discussed. Here, we study breakdowns triggered by failures of links and overloads in networks with a spatial characteristic link length $ζ$. Our results indicate that this abrupt transition has features and critical exponents similar to those of interdependent networks, suggesting that both systems are in the same universality class. For weakly embedded systems (i.e., $ζ$ of the order of the system size $L$) we observe a mixed-order transition, where the order parameter collapses following a long critical plateau. On the other hand, strongly embedded systems (i.e., $ζ\ll L$) exhibit a pure first-order transition, involving nucleation and the growth of damage. The system's critical behavior in both limits is similar to that observed in interdependent networks.

physics.soc-ph↗

Peak fraction of infected in epidemic spreading for multi-community networks

One of the most effective strategies to mitigate the global spreading of a pandemic (e.g., COVID-19) is to shut down international airports. From a network theory perspective, this is since international airports and flights, essentially playing the roles of bridge nodes and bridge links between countries as individual communities, dominate the epidemic spreading characteristics in the whole multi-community system. Among all epidemic characteristics, the peak fraction of infected, $I_{\max}$, is a decisive factor in evaluating an epidemic strategy given limited capacity of medical resources, but is seldom considered in multi-community models. In this paper, we study a general two-community system interconnected by a fraction $r$ of bridge nodes and its dynamic properties, especially $I_{\max}$, under the evolution of the Susceptible-Infected-Recovered (SIR) model. Comparing the characteristic time scales of different parts of the system allows us to analytically derive the asymptotic behavior of $I_{\max}$ with $r$, as $r\rightarrow 0$, which follows different power-law relations in each regime of the phase diagram. We also detect crossovers when $I_{\max}$ changes from one power law to another, crossing different power-law regimes as driven by $r$. Our results enable a better prediction of the effectiveness of strategies acting on bridge nodes, denoted by the power-law exponent $ε_I$ as in $I_{\max}\propto r^{1/ε_I}$.

physics.soc-ph↗

An epidemic model for COVID-19 transmission in Argentina: Exploration of the alternating quarantine and massive testing strategies

The COVID-19 pandemic has challenged authorities at different levels of government administration around the globe. When faced with diseases of this severity, it is useful for the authorities to have prediction tools to estimate in advance the impact on the health system and the human, material, and economic resources that will be necessary. In this paper, we construct an extended Susceptible-Exposed-Infected-Recovered model that incorporates the social structure of Mar del Plata, the $4^\circ$ most inhabited city in Argentina and head of the Municipality of General Pueyrredón. Moreover, we consider detailed partitions of infected individuals according to the illness severity, as well as data of local health resources, to bring these predictions closer to the local reality. Tuning the corresponding epidemic parameters for COVID-19, we study an alternating quarantine strategy, in which a part of the population can circulate without restrictions at any time, while the rest is equally divided into two groups and goes on successive periods of normal activity and lockdown, each one with a duration of $τ$ days. Besides, we implement a random testing strategy over the population. We found that $τ= 7$ is a good choice for the quarantine strategy since it matches with the weekly cycle as it reduces the infected population. Focusing on the health system, projecting from the situation as of September 30, we foresee a difficulty to avoid saturation of ICU, given the extremely low levels of mobility that would be required. In the worst case, our model estimates that four thousand deaths would occur, of which 30\% could be avoided with proper medical attention. Nonetheless, we found that aggressive testing would allow an increase in the percentage of people that can circulate without restrictions, being the equipment required to deal with the additional critical patients relatively low.

q-bio.PE↗

Cascading failures in anisotropic interdependent networks of spatial modular structures

The structure of real-world multilayer infrastructure systems usually exhibits anisotropy due to constraints of the embedding space. For example, geographical features like mountains, rivers and shores influence the architecture of critical infrastructure networks. Moreover, such spatial networks are often non-homogeneous but rather have a modular structure with dense connections within communities and sparse connections between neighboring communities. When the networks of the different layers are interdependent, local failures and attacks may propagate throughout the system. Here we study the robustness of spatial interdependent networks which are both anisotropic and heterogeneous. We also evaluate the effect of localized attacks having different geometrical shapes. We find that anisotropic networks are more robust against localized attacks and that anisotropic attacks, surprisingly, even on isotropic structures, are more effective than isotropic attacks.

physics.soc-ph↗

Role of bridge nodes in epidemic spreading: Different regimes and crossovers

Power-law behaviors are common in many disciplines, especially in network science. Real-world networks, like disease spreading among people, are more likely to be interconnected communities, and show richer power-law behaviors than isolated networks. In this paper, we look at the system of two communities which are connected by bridge links between a fraction $r$ of bridge nodes, and study the effect of bridge nodes to the final state of the Susceptible-Infected-Recovered model, by mapping it to link percolation. By keeping a fixed average connectivity, but allowing different transmissibilities along internal and bridge links, we theoretically derive different power-law asymptotic behaviors of the total fraction of the recovered $R$ in the final state as $r$ goes to zero, for different combinations of internal and bridge link transmissibilities. We also find crossover points where $R$ follows different power-law behaviors with $r$ on both sides when the internal transmissibility is below but close to its critical value, for different bridge link transmissibilities. All of these power-law behaviors can be explained through different mechanisms of how finite clusters in each community are connected into the giant component of the whole system, and enable us to pick effective epidemic strategies and to better predict their impacts.

physics.soc-ph↗

Cascading Failures in Complex Networks

Cascading failure is a potentially devastating process that spreads on real-world complex networks and can impact the integrity of wide-ranging infrastructures, natural systems, and societal cohesiveness. One of the essential features that create complex network vulnerability to failure propagation is the dependency among their components, exposing entire systems to significant risks from destabilizing hazards such as human attacks, natural disasters or internal breakdowns. Developing realistic models for cascading failures as well as strategies to halt and mitigate the failure propagation can point to new approaches to restoring and strengthening real-world networks. In this review, we summarize recent progress on models developed based on physics and complex network science to understand the mechanisms, dynamics and overall impact of cascading failures. We present models for cascading failures in single networks and interdependent networks and explain how different dynamic propagation mechanisms can lead to an abrupt collapse and a rich dynamic behavior. Finally, we close the review with novel emerging strategies for containing cascades of failures and discuss open questions that remain to be addressed.

physics.soc-ph↗

Controlling distant contacts to reduce disease spreading on disordered complex networks

In real social networks, person-to-person interactions are known to be heterogeneous, which can affect the way a disease spreads through a population, reaches a tipping point in the fraction of infected individuals, and becomes an epidemic. This property, called disorder, is usually associated with contact times between individuals and can be modeled by a weighted network, where the weights are related to normalized contact times $ω$. In this paper, we study the SIR model for disease spreading when both close and distant types of interactions are present. We develop a mitigation strategy that reduces only the time duration of distant contacts, which are easier to alter in practice. Using branching theory, supported by simulations, we found that the effectiveness of the strategy increases when the density $f_1$ of close contacts decreases. Moreover, we found a threshold $\tilde{f}_1 = T_c / β$ below which the strategy can bring the system from an epidemic to a non-epidemic phase, even when close contacts have the longest time durations.

physics.soc-ph↗

On the growth of non-motile bacteria colonies: an agent-based model for pattern formation

In the growth of bacterial colonies, a great variety of complex patterns are observed in experiments, depending on external conditions and the bacterial species. Typically, existing models employ systems of reaction-diffusion equations or consist of growth processes based on rules, and are limited to a discrete lattice. In contrast, the two-dimensional model proposed here is an off-lattice simulation, where bacteria are modelled as rigid circles and nutrients are point-like, Brownian particles. Varying the nutrient diffusion and concentration, we simulate a wide range of morphologies compatible with experimental observations, from round and compact to extremely branched patterns. A scaling relationship is found between the number of cells in the interface and the total number of cells, with two characteristic regimes. These regimes correspond to the compact and branched patterns, which are exhibited for sufficiently small and large colonies, respectively. In addition, we characterise the screening effect observed in the structures by analysing the multifractal properties of the growth probability.

physics.bio-ph↗

Containing misinformation spreading in temporal social networks

Many researchers from a variety of fields including computer science, network science and mathematics have focused on how to contain the outbreaks of Internet misinformation that threaten social systems and undermine societal health. Most research on this topic treats the connections among individuals as static, but these connections change in time, and thus social networks are also temporal networks. Currently there is no theoretical approach to the problem of containing misinformation outbreaks in temporal networks. We thus propose a misinformation spreading model for temporal networks and describe it using a new theoretical approach. We propose a heuristic-containing (HC) strategy based on optimizing final outbreak size that outperforms simplified strategies such as those that are random-containing (RC) and targeted-containing (TC). We verify the effectiveness of our HC strategy on both artificial and real-world networks by performing extensive numerical simulations and theoretical analyses. We find that the HC strategy greatly increases the outbreak threshold and decreases the final outbreak threshold.

physics.soc-ph↗

Social contagions with communication channels alternation on multiplex networks

Internet communication channels, e.g., Facebook, Twitter, and email, are multiplex networks that facilitate interaction and information-sharing among individuals. During brief time periods users often use a single communication channel, but then communication channel alteration (CCA) occurs. This means that we must refine our understanding of the dynamics of social contagions. We propose a non-Markovian behavior spreading model in multiplex networks that takes into account the CCA mechanism, and we develop a generalized edge-based compartmental method to describe the spreading dynamics. Through extensive numerical simulations and theoretical analyses we find that the time delays induced by CCA slow the behavior spreading but do not affect the final adoption size. We also find that the CCA suppresses behavior spreading. On two coupled random regular networks, the adoption size exhibits hybrid growth, i.e., it grows first continuously and then discontinuously with the information transmission probability. CCA in ER-SF multiplex networks in which two subnetworks are Erdős-Rényi (ER) and scale-free (SF) introduces a crossover from continuous to hybrid growth in adoption size versus information transmission probability. Our results extend our understanding of the role of CCA in spreading dynamics, and may elicit further research.

physics.soc-ph↗

Strategy for stopping failure cascades in interdependent networks

Interdependencies are ubiquitous throughout the world. Every real-world system interacts with and is dependent on other systems, and this interdependency affects their performance. In particular, interdependencies among networks make them vulnerable to failure cascades, the effects of which are often catastrophic. Failure propagation fragments network components, disconnects them, and may cause complete systemic failure. We propose a strategy of avoiding or at least mitigating the complete destruction of a system of interdependent networks experiencing a failure cascade. Starting with a fraction $1-p$ of failing nodes in one network, we reconnect with a probability $γ$ every isolated component to a functional giant component (GC), the largest connected cluster. We find that as $γ$ increases the resilience of the system to cascading failure also increases. We also find that our strategy is more effective when it is applied in a network of low average degree. We solve the problem theoretically using percolation theory, and we find that the solution agrees with simulation results.

physics.soc-ph↗

Optimal community structure for social contagions

Community structure is an important factor in the behavior of real-world networks because it strongly affects the stability and thus the phase transition order of the spreading dynamics. We here propose a reversible social contagion model of community networks that includes the factor of social reinforcement. In our model an individual adopts a social contagion when the number of received units of information exceeds its adoption threshold. We use mean-field approximation to describe our proposed model, and the results agree with numerical simulations. The numerical simulations and theoretical analyses both indicate that there is a first-order phase transition in the spreading dynamics, and that a hysteresis loop emerges in the system when there is a variety of initially-adopted seeds. We find an optimal community structure that maximizes spreading dynamics. We also find a rich phase diagram with a triple point that separates the no-diffusion phase from the two diffusion phases.

physics.soc-ph↗

Optimal resource diffusion for suppressing disease spreading in multiplex networks

Resource diffusion is an ubiquitous phenomenon, but how it impacts epidemic spreading has received little study. We propose a model that couples epidemic spreading and resource diffusion in multiplex networks. The spread of disease in a physical contact layer and the recovery of the infected nodes are both strongly dependent upon resources supplied by their counterparts in the social layer. The generation and diffusion of resources in the social layer are in turn strongly dependent upon the state of the nodes in the physical contact layer. Resources diffuse preferentially or randomly in this model. To quantify the degree of preferential diffusion, a bias parameter that controls the resource diffusion is proposed. We conduct extensive simulations and find that the preferential resource diffusion can change phase transition type of the fraction of infected nodes. When the degree of interlayer correlation is below a critical value, increasing the bias parameter changes the phase transition from double continuous to single continuous. When the degree of interlayer correlation is above a critical value, the phase transition changes from multiple continuous to first discontinuous and then to hybrid. We find hysteresis loops in the phase transition. We also find that there is an optimal resource strategy at each fixed degree of interlayer correlation where the threshold reaches a maximum and under which the disease can be maximally suppressed. In addition, the optimal controlling parameter increases as the degree of inter-layer correlation increases.

physics.soc-ph↗

Suppressing epidemic spreading in multiplex networks with social-support

Although suppressing the spread of a disease is usually achieved by investing in public resources, in the real world only a small percentage of the population have access to government assistance when there is an outbreak, and most must rely on resources from family or friends. We study the dynamics of disease spreading in social-contact multiplex networks when the recovery of infected nodes depends on resources from healthy neighbors in the social layer. We investigate how degree heterogeneity affects the spreading dynamics. Using theoretical analysis and simulations we find that degree heterogeneity promotes disease spreading. The phase transition of the infected density is hybrid and increases smoothly from zero to a finite small value at the first invasion threshold and then suddenly jumps at the second invasion threshold. We also find a hysteresis loop in the transition of the infected density. We further investigate how an overlap in the edges between two layers affects the spreading dynamics. We find that when the amount of overlap is smaller than a critical value the phase transition is hybrid and there is a hysteresis loop, otherwise the phase transition is continuous and the hysteresis loop vanishes. In addition, the edge overlap allows an epidemic outbreak when the transmission rate is below the first invasion threshold, but suppresses any explosive transition when the transmission rate is above the first invasion threshold.

physics.soc-ph↗

Promoting information spreading by using contact memory

Promoting information spreading is a booming research topic in network science community. However, the exiting studies about promoting information spreading seldom took into account the human memory, which plays an important role in the spreading dynamics. In this paper we propose a non-Markovian information spreading model on complex networks, in which every informed node contacts a neighbor by using the memory of neighbor's accumulated contact numbers in the past. We systematically study the information spreading dynamics on uncorrelated configuration networks and a group of $22$ real-world networks, and find an effective contact strategy of promoting information spreading, i.e., the informed nodes preferentially contact neighbors with small number of accumulated contacts. According to the effective contact strategy, the high degree nodes are more likely to be chosen as the contacted neighbors in the early stage of the spreading, while in the late stage of the dynamics, the nodes with small degrees are preferentially contacted. We also propose a mean-field theory to describe our model, which qualitatively agrees well with the stochastic simulations on both artificial and real-world networks.

physics.soc-ph↗

Unification of theoretical approaches for epidemic spreading on complex networks

Models of epidemic spreading on complex networks have attracted great attention among researchers in physics, mathematics, and epidemiology due to their success in predicting and controlling scenarios of epidemic spreading in real-world scenarios. To understand the interplay between epidemic spreading and the topology of a contact network, several outstanding theoretical approaches have been developed. An accurate theoretical approach describing the spreading dynamics must take both the network topology and dynamical correlations into consideration at the expense of increasing the complexity of the equations. In this short survey we unify the most widely used theoretical approaches for epidemic spreading on complex networks in terms of increasing complexity, including the mean-field, the heterogeneous mean-field, the quench mean-field, dynamical message-passing, link percolation, and pairwise approximation. We build connections among these approaches to provide new insights into developing an accurate theoretical approach to spreading dynamics on complex networks.

physics.soc-ph↗

Fluctuations of a surface relaxation model in interacting scale free networks

Isolated complex networks have been studied deeply in the last decades due to the fact that many real systems can be modeled using these types of structures. However, it is well known that the behavior of a system not only depends on itself, but usually also depends on the dynamics of other structures. For this reason, interacting complex networks and the processes developed on them have been the focus of study of many researches in the last years. One of the most studied subjects in this type of structures is the Synchronization problem, which is important in a wide variety of processes in real systems. In this manuscript we study the synchronization of two interacting scale-free networks, in which each node has $ke$ dependency links with different nodes in the other network. We map the synchronization problem with an interface growth, by studying the fluctuations in the steady state of a scalar field defined in both networks. We find that as $ke$ slightly increases from $ke=0$, there is a really significant decreasing in the fluctuations of the system. However, this considerable improvement takes place mainly for small values of $ke$, when the interaction between networks becomes stronger there is only a slight change in the fluctuations. We characterize how the dispersion of the scalar field depends on the internal degree, and we show that a combination between the decreasing of this dispersion and the integer nature of our growth model are the responsible for the behavior of the fluctuations with $ke$.

physics.soc-ph↗

Suppressing disease spreading by using information diffusion on multiplex networks

Although there is always an interplay between the dynamics of information diffusion and disease spreading, the empirical research on the systemic coevolution mechanisms connecting these two spreading dynamics is still lacking. Here we investigate the coevolution mechanisms and dynamics between information and disease spreading by utilizing real data and a proposed spreading model on multiplex network. Our empirical analysis finds asymmetrical interactions between the information and disease spreading dynamics. Our results obtained from both the theoretical framework and extensive stochastic numerical simulations suggest that an information outbreak can be triggered in a communication network by its own spreading dynamics or by a disease outbreak on a contact network, but that the disease threshold is not affected by information spreading. Our key finding is that there is an optimal information transmission rate that markedly suppresses the disease spreading. We find that the time evolution of the dynamics in the proposed model qualitatively agrees with the real-world spreading processes at the optimal information transmission rate.

physics.soc-ph↗