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M. A. Di Muro

Publications and source records attributed to M. A. Di Muro.

12 recordsLinked to original sources

Viscosity as the product of its ideal low-concentration value times a thermodynamic function

The behavior of viscosity, $η$, as a function of concentration in dense fluids remains an unsolved problem, as is the case with other transport coefficients. Boltzmann's theory and the Chapman-Enskog method predict the value of the viscosity at low concentrations, $η_0$. Here, the hypothesis $η=ϕ\, η_0$ is proposed, where $ϕ$ is a function of the thermodynamic state that represents the effects of interactions as concentration increases. We consider that $η_0$ is the viscosity in an ideal hypothetical system, where the condition of small interactions applies for the whole density range ($ϕ\to 1$ for low concentration). The method proposed to verify this hypothesis involves coupling the system with a solvent represented by a Langevin thermostat, characterized by a damping time $t_d$. Molecular dynamics simulations show that different values of noise intensity modify $η$ and $η_0$, but do not affect $ϕ$. This result supports the assumption that $ϕ$ is a state function, since the thermodynamic state remains unaltered by the presence of damping and noise. Simulations were conducted for particles that interact via a pseudo-hard sphere or a Lennard-Jones potential.

cond-mat.stat-mech↗

Dependence on the thermodynamic state of self-diffusion of pseudo hard-spheres

Self-diffusion, $D$, in a system of particles that interact with a pseudo hard sphere potential is analyzed. Coupling with a solvent is represented by a Langevin thermostat, characterized by the damping time $t_d$. The hypotheses that $D=D_0 φ$ is proposed, where $D_0$ is the small concentration diffusivity and $φ$ is a thermodynamic function that represents the effects of interactions as concentration is increased. Molecular dynamics simulations show that different values of the noise intensity modify $D_0$ but do not modify $φ$. This result is consistent with the assumption that $φ$ is a thermodynamic function, since the thermodynamic state is not modified by the presence of damping and noise.

cond-mat.stat-mech↗

Disease spreading with social distancing: A prevention strategy in disordered multiplex networks

The frequent emergence of diseases with the potential to become threats at local and global scales, such as influenza A(H1N1), SARS, MERS, and recently COVID-19 disease, makes it crucial to keep designing models of disease propagation and strategies to prevent or mitigate their effects in populations. Since isolated systems are exceptionally rare to find in any context, especially in human contact networks, here we examine the susceptible-infected-recovered model of disease spreading in a multiplex network formed by two distinct networks or layers, interconnected through a fraction $q$ of shared individuals (overlap). We model the interactions through weighted networks, because person-to-person interactions are diverse (or disordered); weights represent the contact times of the interactions. Using branching theory supported by simulations, we analyze a social distancing strategy that reduces the average contact time in both layers, where the intensity of the distancing is related to the topology of the layers. We find that the critical values of the distancing intensities, above which an epidemic can be prevented, increase with the overlap $q$. Also we study the effect of the social distancing on the mutual giant component of susceptible individuals, which is crucial to keep the functionality of the system. In addition, we find that for relatively small values of the overlap $q$, social distancing policies might not be needed at all to maintain the functionality of the system.

physics.soc-ph↗

Reversible bootstrap percolation: Fake news and fact checking

Bootstrap percolation has been used to describe opinion formation in society and other social and natural phenomena. The formal equation of the bootstrap percolation may have more than one solution, corresponding to several stable fixed points of the corresponding iteration process. We construct a reversible bootstrap percolation process, which converges to these extra solutions displaying a hysteresis typical of discontinuous phase transitions. This process provides a reasonable model for fake news spreading and the effectiveness of fact checking. We show that sometimes it is not sufficient to discard all the sources of fake news in order to reverse the belief of a population that formed under the influence of these sources.

physics.soc-ph↗

Insights into bootstrap percolation: Its equivalence with k-core percolation and the giant component

K-core and bootstrap percolation are widely studied models that have been used to represent and understand diverse deactivation and activation processes in natural and social systems. Since these models are considerably similar, it has been suggested in recent years that they could be complementary. In this manuscript we provide a rigorous analysis that shows that for any degree and threshold distributions heterogeneous bootstrap percolation can be mapped into heterogeneous k-core percolation and vice versa, if the functionality thresholds in both processes satisfy a complementary relation. Another interesting problem in bootstrap and k-core percolation is the fraction of nodes belonging to their giant connected components $P_{\infty b}$ and $P_{\infty c}$, respectively. We solve this problem analytically for arbitrary randomly connected graphs and arbitrary threshold distributions, and we show that $P_{\infty b}$ and $P_{\infty c}$ are not complementary. Our theoretical results coincide with computer simulations in the limit of very large graphs. In bootstrap percolation, we show that when using the branching theory to compute the size of the giant component, we must consider two different types of links, which are related to distinct spanning branches of active nodes.

physics.soc-ph↗

Dynamic vaccination in partially overlapped multiplex network

In this work we propose and investigate a new strategy of vaccination, which we call "dynamic vaccination". In our model, susceptible people become aware that one or more of their contacts are infected, and thereby get vaccinated with probability $ω$, before having physical contact with any infected patient. Then, the non-vaccinated individuals will be infected with probability $β$. We apply the strategy to the SIR epidemic model in a multiplex network composed by two networks, where a fraction $q$ of the nodes acts in both networks. We map this model of dynamic vaccination into bond percolation model, and use the generating functions framework to predict theoretically the behavior of the relevant magnitudes of the system at the steady state. We find a perfect agreement between the solutions of the theoretical equations and the results of stochastic simulations. In addition, we find an interesting phase diagram in the plane $β-ω$, which is composed by an epidemic and a non-epidemic phases, separated by a critical threshold line $β_c$, which depends on $q$. Wefind that, for all values of $q$, a region in the diagram where the vaccination is so efficient that, regardless of the virulence of the disease, it never becomes an epidemic. We compare our strategy with random immunization and find that using the same amount of vaccines for both scenarios, we obtain that the spread of the disease is much lower in the case of dynamic vaccination when compared to random immunization. Furthermore, we also compare our strategy with targeted immunization and we find that, depending on $ω$, dynamic vaccination will perform significantly better, and in some cases will stop the disease before it becomes an epidemic.

physics.soc-ph↗

Multiple outbreaks in epidemic spreading with local vaccination and limited vaccines

How to prevent the spread of human diseases is a great challenge for the scientific community and so far there are many studies in which immunization strategies have been developed. However, these kind of strategies usually do not consider that medical institutes may have limited vaccine resources available. In this manuscript, we explore the Susceptible-Infected-Recovered (SIR) model with local dynamic vaccination, and considering limited vaccines. In this model, susceptibles in contact with an infected individual, are vaccinated -with probability $ω$- and then get infected -with probability $β$. However, when the fraction of immunized individuals reaches a threshold $V_L$, the vaccination stops, after which only the infection is possible. In the steady state, besides the critical points $β_c$ and $ω_c$ that separate a non-epidemic from an epidemic phase, we find for a range of $V_L$ another transition points, $β^*>β_c$ and $ω^*<ω_c$, which correspond to a novel discontinuous phase transition. This critical value separates a phase where the amount of vaccines is sufficient, from a phase where the disease is strong enough to exhaust all the vaccination units. For a disease with fixed $β$, the vaccination probability $ω$ can be controlled in order to drastically reduce the number of infected individuals, using efficiently the available vaccines. Furthermore, the temporal evolution of the system close to $β^*$ or $ω^*$, shows that after a peak of infection the system enters into a quasi-stationary state, with only a few infected cases. But if there are no more vaccines, these few infected individuals could originate a second outbreak, represented by a second peak of infection. This state of apparent calm, could be dangerous since it may lead to misleading conclusions and to an abandon of the strategies to control the disease.

physics.soc-ph↗

Cascading Failures in Interdependent Networks with Multiple Supply-Demand Links and Functionality Thresholds

Various social, financial, biological and technological systems can be modeled by interdependent networks. It has been assumed that in order to remain functional, nodes in one network must receive the support from nodes belonging to different networks. So far these models have been limited to the case in which the failure propagates across networks only if the nodes lose all their supply nodes. In this paper we develop a more realistic model for two interdependent networks in which each node has its own supply threshold, i.e., they need the support of a minimum number of supply nodes to remain functional. In addition, we analyze different conditions of internal node failure due to disconnection from nodes within its own network. We show that several local internal failure conditions lead to similar nontrivial results. When there are no internal failures the model is equivalent to a bipartite system, which can be useful to model a financial market. We explore the rich behaviors of these models that include discontinuous and continuous phase transitions. Using the generating functions formalism, we analytically solve all the models in the limit of infinitely large networks and find an excellent agreement with the stochastic simulations.

physics.soc-ph↗

Cascading failures in interdependent networks with finite functional components

We present a cascading failure model of two interdependent networks in which functional nodes belong to components of size greater than or equal to $s$. We find theoretically and via simulation that in complex networks with random dependency links the transition is first-order for $s\geq 3$ and second-order for $s=2$. We find for two square lattices with a distance constraint $r$ in the dependency links that increasing $r$ moves the system from a regime without a phase transition to one with a second-order transition. As $r$ continues to increase the system collapses in a first-order transition. Each regime is associated with a different structure of domain formation of functional nodes.

physics.soc-ph↗

Failure-recovery model with competition between failures in complex networks: a dynamical approach

Real systems are usually composed by units or nodes whose activity can be interrupted and restored intermittently due to complex interactions not only with the environment, but also with the same system. Majdandžić $et\;al.$ [Nature Physics 10, 34 (2014)] proposed a model to study systems in which active nodes fail and recover spontaneously in a complex network and found that in the steady state the density of active nodes can exhibit an abrupt transition and hysteresis depending on the values of the parameters. Here we investigate a model of recovery-failure from a dynamical point of view. Using an effective degree approach we find that the systems can exhibit a temporal sharp decrease in the fraction of active nodes. Moreover we show that, depending on the values of the parameters, the fraction of active nodes has an oscillatory regime which we explain as a competition between different failure processes. We also find that in the non-oscillatory regime, the critical fraction of active nodes presents a discontinuous drop which can be related to a "targeted" k-core percolation process. Finally, using mean field equations we analyze the space of parameters at which hysteresis and oscillatory regimes can be found.

physics.soc-ph↗

Synchronization in interacting Scale Free Networks

We study the fluctuations of the interface, in the steady state, of the Surface Relaxation Model (SRM) in two scale free interacting networks where a fraction $q$ of nodes in both networks interact one to one through external connections. We find that as $q$ increases the fluctuations on both networks decrease and thus the synchronization reaches an improvement of nearly $40\%$ when $q=1$. The decrease of the fluctuations on both networks is due mainly to the diffusion through external connections which allows to reducing the load in nodes by sending their excess mostly to low-degree nodes, which we report have the lowest heights. This effect enhances the matching of the heights of low-and high-degree nodes as $q$ increases reducing the fluctuations. This effect is almost independent of the degree distribution of the networks which means that the interconnection governs the behavior of the process over its topology.

physics.soc-ph↗

Recovery of Interdependent Networks

Recent network research has focused on the cascading failures in a system of interdependent networks and the necessary preconditions for system collapse. An important question that has not been addressed is how to repair a failing system before it suffers total breakdown. Here we introduce a recovery strategy of nodes and develop an analytic and numerical framework for studying the concurrent failure and recovery of a system of interdependent networks based on an efficient and practically reasonable strategy. Our strategy consists of repairing a fraction of failed nodes, with probability of recovery $γ$, that are neighbors of the largest connected component of each constituent network. We find that, for a given initial failure of a fraction $1-p$ of nodes, there is a critical probability of recovery above which the cascade is halted and the system fully restores to its initial state and below which the system abruptly collapses. As a consequence we find in the plane $γ-p$ of the phase diagram three distinct phases. A phase in which the system never collapses without being restored, another phase in which the recovery strategy avoids the breakdown, and a phase in which even the repairing process cannot avoid the system collapse.

physics.soc-ph↗