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Meiling Xie

Publications and source records attributed to Meiling Xie.

2 recordsLinked to original sources

Adaptive Epidemic Dynamics on Hypergraphs with Group-Level Immunization and Rewiring

Understanding how higher-order social structures shape epidemic spreading requires models that couple group interactions with adaptive behavior. We introduce an adaptive simplicial susceptible-infected-susceptible (s-SIS) model on d-uniform hypergraphs, where both node states and hyperedge activity co-evolve in response to local infection pressure. Hyperedges represent group interactions of fixed size and dynamically reduce their activity through a feedback mechanism in highly infected environments. Within this framework, we design two classes of hyperedge-level interventions: (i) risk-driven immunization, combining spontaneous, activity-based isolation with targeted deactivation guided by hyperedge infection pressure, and (ii) structural rewiring, which reconstructs group structures either randomly or via degree-preferential attachment. By extending the microscopic Markov chain approximation to higher-order interactions, we derive analytical conditions for the existence and stability of both endemic and disease-free stationary states. Our analysis shows that adaptive hyperedge feedback can induce discontinuous phase transitions, nonlinear epidemic thresholds, and bistable regimes in which sufficiently high initial prevalence drives the system to a disease-free equilibrium. Extensive Monte Carlo simulations support the theory and confirm that targeted immunization and degree-preferential rewiring substantially suppress epidemic prevalence, outperforming random strategies. These results demonstrate that higher-order interactions and adaptive group-level responses fundamentally reshape epidemic bifurcations and suggest principles for designing effective intervention policies in complex social systems.

physics.soc-ph

SIS Epidemic Modelling on Homogeneous Networked System: General Recovering Process and Mean-Field Perspective

Although we have made progress in understanding disease spread in complex systems with non-Poissonian activity patterns, current models still fail to capture the full range of recovery time distributions. In this paper, we propose an extension of the classic susceptible-infected-susceptible (SIS) model, called the general recovering process SIS (grp-SIS) model. This model incorporates arbitrary recovery time distributions for infected nodes within the system. We derive the mean-field equations assuming a homogeneous network, provide solutions for specific recovery time distributions, and investigate the probability density function (PDF) for infection times in the system's steady state. Our findings show that recovery time distributions significantly affect disease dynamics, and we suggest several future research directions, including extending the model to arbitrary infection processes and using the quasistationary method to address deviations in numerical results.

cs.SI