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Peter Kissack

Publications and source records attributed to Peter Kissack.

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Evaluating Deep Multivariate Imputation Models on Wearable Device Data

Wearable device data enables continuous health monitoring, but suffers from structured missingness: features sharing a physical sensor drop out together. Deep imputation methods such as BRITS and SAITS have seen limited evaluation on multimodal physiological data under realistic missingness, and existing benchmarks use random-point holdout protocols that incorrectly assume missingness is independent across features and time. Using data from a person with epilepsy recorded on a Garmin smartwatch, we develop an evaluation protocol that mines contiguous missing-run templates from training data, stratifies them by per-feature gap-length quantiles, and injects them as block masks with preserved co-missingness structure. A matched training protocol exposing models to the same missingness distribution reduces BRITS's severe-gap MAE by 43%, demonstrating the potential benefit of the proposed evaluation and training protocol within this single-participant dataset. We further extend BRITS with time-of-day encoding and a circadian harmonic channel. No single model dominates: linear interpolation is optimal for slow-moving features over short gaps; extended BRITS achieves lower MAE on dynamic cardiac features in moderate and severe gaps; and SAITS better preserves the ground-truth distribution by Jensen-Shannon distance despite higher MAE. Ultimately, model rankings strongly depend on evaluation designs. By exposing how traditional evaluation methods obscure true model capabilities, our transferable protocol establishes critical steps towards developing better imputation strategies for future multi-sensor wearable datasets.

cs.LG

Trophic structure predicts seizure propagation in brain network models

Epilepsy is widely regarded as a disorder driven by connectivity in the brain. We use a model of seizure dynamics on directed networks to investigate how structural properties affect seizure propensity. We find that properties such as trophic coherence, spectral radius, strong connectivity and non-normality are closely related to seizure propensity, and present a proof of a theoretical relationship between spectral radius and cycle structure. Our simulated results are robust to the kind of coupling used in the model and become stronger as network size is increased. They suggest that the overall directionality of information processing in the brain may be related to a propensity for epileptic seizures.

q-bio.NC