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R. Shashangan

Publications and source records attributed to R. Shashangan.

3 recordsLinked to original sources

Controlling extreme events in neuronal networks: A single driving signal approach

We show that in a drive-response coupling framework extreme events are suppressed in the response system by the dominance of a single driving signal. We validate this approach across three distinct response network topologies, namely (i) a pair of coupled neurons, (ii) a monolayer network of N coupled neurons and (iii) a two-layer multiplex network each composed of FitzHugh-Nagumo neuronal units. The response networks inherently exhibit extreme events. Our results demonstrate that influencing just one neuron in the response network with an appropriately tuned driving signal is sufficient to control extreme events across all three configurations. In the two-neuron case, suppression of extreme events occurs due to the breaking of phase-locking between the driving neuron and the targeted response neuron. In the case of monolayer and multiplex networks, suppression of extreme events results from the disruption of protoevent frequency dynamics and a subsequent frequency decoupling of the driven neuron from the rest of the network. We also observe that when the size of the neurons in response network connected to the drive increases, the onset of control occurs earlier indicating a scaling advantage of the method.

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Propagation of extreme events in multiplex neuronal networks

In previous studies, the propagation of extreme events across nodes in monolayer networks has been extensively studied. In this work, we extend this investigation to explore the propagation of extreme events between two distinct layers in a multiplex network. We consider a two-layer network, where one layer is globally coupled and exhibits extreme events, while the second layer remains uncoupled. The interlayer connections between the layers are either unidirectional or bidirectional. We find that unidirectional coupling between the layers can induce extreme events in the uncoupled layer, whereas bidirectional coupling tends to mitigate extreme events in the globally coupled layer. To characterize extreme and non-extreme states, we use probability plots to identify distinct regions in the parameter space. Additionally, we study the robustness of extreme events emergence by examining various network topologies in the uncoupled layer. The mechanism behind the occurrence of extreme events is explored, with a particular focus on the transition from asynchronous states to a fully synchronized excitable state. For numerical simulations, we use nonidentical FitzHugh-Nagumo neurons at each node, which captures the dynamical behavior of both coupled and uncoupled layers. Our findings suggest that extreme events in the uncoupled layer emerge through the gradual disappearance of disorder, accompanied by occasional bursts of synchronized activity. Results obtained in this work will serve a starting point in understanding the dynamics behind the propagation of extreme events in real-world networks.

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Mitigation of extreme events in an excitable system

Formulating mitigation strategies is one of the main aspect in the dynamical study of extreme events. Apart from the effective control, easy implementation of the devised tool should also be given importance. In this work, we analyze the mitigation of extreme events in a coupled FitzHugh-Nagumo (FHN) neuron model utilizing an easily implementable constant bias analogous to a constant DC stimulant. We report the route through which the extreme events gets mitigated in $Two$, $Three$ and $N-$coupled FHN systems. In all the three cases, extreme events in the observable $\bar{x}$ gets suppressed. We confirm our results with the probability distribution function of peaks, $d_{max}$ plot and probability plots. Here $d_{max}$ is a measure of number of standard deviations that crosses the average amplitude corresponding to $\bar{x}_{max}$. Interestingly, we found that constant bias suppresses the extreme events without changing the collective frequency of the system.

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