arXiv subjects

Tristan Manfred Stöber

Publications and source records attributed to Tristan Manfred Stöber.

2 recordsLinked to original sources

arXiv2026

FUBU-EPSTEIN: A Large-Scale Twitter Dataset on the Jeffrey Epstein Case and Its Global Public Discourse (2019-2023)

The criminal case of Jeffrey Epstein has generated a complex, long-running global discourse on digital platforms, characterized by punctuated attention shocks, conspiracy theories, and blame attribution. To facilitate the computational study of these dynamics, we introduce the FUBU-EPSTEIN dataset, a large-scale, multi-dimensional research corpus of 54.38 million Twitter statuses authored by 7.11 million users and collected between August 2019 and April 2023. The source corpus was captured continuously in near-real time and enriched with a directed social contact graph of 37.03 million edge rows and annotations from Qwen2.5-7B-Instruct covering sentiment, conspiracy and misinformation stance, toxicity, moral emotion, and related dimensions. For public distribution, we created a textless, de-identified derivative that retains one row for every deduplicated status, categorical and numeric annotations, coarse temporal information, and 46.15 million internal status relationships. It excludes tweet text, original post and user identifiers, handles, profile fields, exact timestamps, and reverse mappings. The resulting release supports longitudinal content and diffusion analyses while reducing disclosure and platform-content redistribution risks. To request access to raw data for collaborative research under ethical and legal safeguards, contact fubu.dataset@gmail.com.

cs.SI↗

arXiv2022

Degeneracy in epilepsy: Multiple Routes to Hyperexcitable Brain Circuits and their Repair

Developing effective therapies against epilepsy remains a challenge. The complex and multifaceted nature of this disease still fuels controversies about its origin. In this perspective article, we argue that conflicting hypotheses can be reconciled by taking into account the degeneracy of the brain, which manifests in multiple routes leading to similar function or dysfunction. We exemplify degeneracy at three different levels, ranging from the cellular to the network and systems level. First, at the cellular level, we describe the relevance of ion channel degeneracy for epilepsy and discuss its interplay with dendritic morphology. Second, at the network level, we provide examples for the degeneracy of synaptic and intrinsic neuronal properties that supports the robustness of neuronal networks but also leads to diverse responses to ictogenic and epileptogenic perturbations. Third, at the system level, we provide examples for degeneracy in the intricate interactions between the immune and nervous system. Finally, we show that computational approaches including multiscale and so called population neural circuit models help disentangle the complex web of physiological and pathological adaptations. Such models may contribute to identifying the best personalized multitarget strategies for directing the system towards a physiological state.

q-bio.NC↗