arXiv subjects

Antoine Allard

Publications and source records attributed to Antoine Allard.

72 records · Page 4Linked to original sources

arXiv2015

Complex networks as an emerging property of hierarchical preferential attachment

Real complex systems are not rigidly structured; no clear rules or blueprints exist for their construction. Yet, amidst their apparent randomness, complex structural properties universally emerge. We propose that an important class of complex systems can be modeled as an organization of many embedded levels (potentially infinite in number), all of them following the same universal growth principle known as preferential attachment. We give examples of such hierarchy in real systems, for instance in the pyramid of production entities of the film industry. More importantly, we show how real complex networks can be interpreted as a projection of our model, from which their scale independence, their clustering, their hierarchy, their fractality and their navigability naturally emerge. Our results suggest that complex networks, viewed as growing systems, can be quite simple, and that the apparent complexity of their structure is largely a reflection of their unobserved hierarchical nature.

physics.soc-ph↗

arXiv2015

A shadowing problem in the detection of overlapping communities: lifting the resolution limit through a cascading procedure

Community detection is the process of assigning nodes and links in significant communities (e.g. clusters, function modules) and its development has led to a better understanding of complex networks. When applied to sizable networks, we argue that most detection algorithms correctly identify prominent communities, but fail to do so across multiple scales. As a result, a significant fraction of the network is left uncharted. We show that this problem stems from larger or denser communities overshadowing smaller or sparser ones, and that this effect accounts for most of the undetected communities and unassigned links. We propose a generic cascading approach to community detection that circumvents the problem. Using real and artificial network datasets with three widely used community detection algorithms, we show how a simple cascading procedure allows for the detection of the missing communities. This work highlights a new detection limit of community structure, and we hope that our approach can inspire better community detection algorithms.

physics.soc-ph↗

arXiv2015

General and exact approach to percolation on random graphs

We present a comprehensive and versatile theoretical framework to study site and bond percolation on clustered and correlated random graphs. Our contribution can be summarized in three main points. (i) We introduce a set of iterative equations that solve the exact distribution of the size and composition of components in finite size quenched or random multitype graphs. (ii) We define a very general random graph ensemble that encompasses most of the models published to this day, and also that permits to model structural properties not yet included in a theoretical framework. Site and bond percolation on this ensemble is solved exactly in the infinite size limit using probability generating functions [i.e., the percolation threshold, the size and the composition of the giant (extensive) and small components]. Several examples and applications are also provided. (iii) Our approach can be adapted to model interdependent graphs---whose most striking feature is the emergence of an extensive component via a discontinuous phase transition---in an equally general fashion. We show how a graph can successively undergo a continuous then a discontinuous phase transition, and preliminary results suggest that clustering increases the amplitude of the discontinuity at the transition.

cond-mat.stat-mech↗

arXiv2015

Prudent behaviour accelerates disease transmission

Infectious diseases often spread faster near their peak than would be predicted given early data on transmission. Despite the commonality of this phenomena, there are no known general mechanisms able to cause an exponentially spreading dis- ease to begin spreading faster. Indeed most features of real world social networks, e.g. clustering1,2 and community structure3, and of human behaviour, e.g. social distancing4 and increased hygiene5, will slow disease spread. Here, we consider a model where individuals with essential societal roles-e.g. teachers, first responders, health-care workers, etc.- who fall ill are replaced with healthy individuals. We refer to this process as relational exchange. Relational exchange is also a behavioural process, but one whose effect on disease transmission is less obvious. By incorporating this behaviour into a dynamic network model, we demonstrate that replacing individuals can accelerate disease transmission. Furthermore, we find that the effects of this process are trivial when considering a standard mass-action model, but dramatic when considering network structure. This result highlights another critical shortcoming in mass-action models, namely their inability to account for behavioural processes. Lastly, using empirical data, we find that this mechanism parsimoniously explains observed patterns across more than seventeen years of influenza and dengue virus data. We anticipate that our findings will advance the emerging field of disease forecasting and will better inform public health decision making during outbreaks.

q-bio.QM↗

arXiv2014

Coexistence of phases and the observability of random graphs

In a recent Letter, Yang et al. [Phys. Rev. Lett. 109, 258701 (2012)] introduced the concept of observability transitions: the percolation-like emergence of a macroscopic observable component in graphs in which the state of a fraction of the nodes, and of their first neighbors, is monitored. We show how their concept of depth-L percolation---where the state of nodes up to a distance L of monitored nodes is known---can be mapped unto multitype random graphs, and use this mapping to exactly solve the observability problem for arbitrary L. We then demonstrate a non-trivial coexistence of an observable and of a non-observable extensive component. This coexistence suggests that monitoring a macroscopic portion of a graph does not prevent a macroscopic event to occur unbeknown to the observer. We also show that real complex systems behave quite differently with regard to observability depending on whether they are geographically-constrained or not.

cond-mat.stat-mech↗

arXiv2013

Percolation on random networks with arbitrary k-core structure

The k-core decomposition of a network has thus far mainly served as a powerful tool for the empirical study of complex networks. We now propose its explicit integration in a theoretical model. We introduce a Hard-core Random Network model that generates maximally random networks with arbitrary degree distribution and arbitrary k-core structure. We then solve exactly the bond percolation problem on the HRN model and produce fast and precise analytical estimates for the corresponding real networks. Extensive comparison with selected databases reveals that our approach performs better than existing models, while requiring less input information.

physics.soc-ph↗

arXiv2013

Global efficiency of local immunization on complex networks

Epidemics occur in all shapes and forms: infections propagating in our sparse sexual networks, rumours and diseases spreading through our much denser social interactions, or viruses circulating on the Internet. With the advent of large databases and efficient analysis algorithms, these processes can be better predicted and controlled. In this study, we use different characteristics of network organization to identify the influential spreaders in 17 empirical networks of diverse nature using 2 epidemic models. We find that a judicious choice of local measures, based either on the network's connectivity at a microscopic scale or on its community structure at a mesoscopic scale, compares favorably to global measures, such as betweenness centrality, in terms of efficiency, practicality and robustness. We also develop an analytical framework that highlights a transition in the characteristic scale of different epidemic regimes. This allows to decide which local measure should govern immunization in a given scenario.

physics.soc-ph↗

arXiv2012

On the constrained growth of complex critical systems

Critical, or scale independent, systems are so ubiquitous, that gaining theoretical insights on their nature and properties has many direct repercussions in social and natural sciences. In this report, we start from the simplest possible growth model for critical systems and deduce constraints in their growth : the well-known preferential attachment principle, and, mainly, a new law of temporal scaling. We then support our scaling law with a number of calculations and simulations of more complex theoretical models : critical percolation, self-organized criticality and fractal growth. Perhaps more importantly, the scaling law is also observed in a number of empirical systems of quite different nature : prose samples, artistic and scientific productivity, citation networks, and the topology of the Internet. We believe that these observations pave the way towards a general and analytical framework for predicting the growth of complex systems.

physics.soc-ph↗

arXiv2012

Bond percolation on a class of correlated and clustered random graphs

We introduce a formalism for computing bond percolation properties of a class of correlated and clustered random graphs. This class of graphs is a generalization of the Configuration Model where nodes of different types are connected via different types of hyperedges, edges that can link more than 2 nodes. We argue that the multitype approach coupled with the use of clustered hyperedges can reproduce a wide spectrum of complex patterns, and thus enhances our capability to model real complex networks. As an illustration of this claim, we use our formalism to highlight unusual behaviors of the size and composition of the components (small and giant) in a synthetic, albeit realistic, social network.

cond-mat.stat-mech↗

arXiv2012

Exact solution of bond percolation on small arbitrary graphs

We introduce a set of iterative equations that exactly solves the size distribution of components on small arbitrary graphs after the random removal of edges. We also demonstrate how these equations can be used to predict the distribution of the node partitions (i.e., the constrained distribution of the size of each component) in undirected graphs. Besides opening the way to the theoretical prediction of percolation on arbitrary graphs of large but finite size, we show how our results find application in graph theory, epidemiology, percolation and fragmentation theory.

cond-mat.stat-mech↗

arXiv2012

Propagation on networks: an exact alternative perspective

By generating the specifics of a network structure only when needed (on-the-fly), we derive a simple stochastic process that exactly models the time evolution of susceptible-infectious dynamics on finite-size networks. The small number of dynamical variables of this birth-death Markov process greatly simplifies analytical calculations. We show how a dual analytical description, treating large scale epidemics with a Gaussian approximations and small outbreaks with a branching process, provides an accurate approximation of the distribution even for rather small networks. The approach also offers important computational advantages and generalizes to a vast class of systems.

cond-mat.stat-mech↗

arXiv2011

Epidemics on contact networks: a general stochastic approach

Dynamics on networks is considered from the perspective of Markov stochastic processes. We partially describe the state of the system through network motifs and infer any missing data using the available information. This versatile approach is especially well adapted for modelling spreading processes and/or population dynamics. In particular, the generality of our systematic framework and the fact that its assumptions are explicitly stated suggests that it could be used as a common ground for comparing existing epidemics models too complex for direct comparison, such as agent-based computer simulations. We provide many examples for the special cases of susceptible-infectious-susceptible (SIS) and susceptible-infectious-removed (SIR) dynamics (e.g., epidemics propagation) and we observe multiple situations where accurate results may be obtained at low computational cost. Our perspective reveals a subtle balance between the complex requirements of a realistic model and its basic assumptions.

physics.soc-ph↗

arXiv2011

Structural preferential attachment: Stochastic process for the growth of scale-free, modular and self-similar systems

Many complex systems have been shown to share universal properties of organization, such as scale independence, modularity and self-similarity. We borrow tools from statistical physics in order to study structural preferential attachment (SPA), a recently proposed growth principle for the emergence of the aforementioned properties. We study the corresponding stochastic process in terms of its time evolution, its asymptotic behavior and the scaling properties of its statistical steady state. Moreover, approximations are introduced to facilitate the modelling of real systems, mainly complex networks, using SPA. Finally, we investigate a particular behavior observed in the stochastic process, the peloton dynamics, and show how it predicts some features of real growing systems using prose samples as an example.

physics.soc-ph↗

arXiv2011

Structural preferential attachment: Network organization beyond the link

We introduce a mechanism which models the emergence of the universal properties of complex networks, such as scale independence, modularity and self-similarity, and unifies them under a scale-free organization beyond the link. This brings a new perspective on network organization where communities, instead of links, are the fundamental building blocks of complex systems. We show how our simple model can reproduce social and information networks by predicting their community structure and more importantly, how their nodes or communities are interconnected, often in a self-similar manner.

physics.soc-ph↗

arXiv2011

Modeling the dynamical interaction between epidemics on overlay networks

Epidemics seldom occur as isolated phenomena. Typically, two or more viral agents spread within the same host population and may interact dynamically with each other. We present a general model where two viral agents interact via an immunity mechanism as they propagate simultaneously on two networks connecting the same set of nodes. Exploiting a correspondence between the propagation dynamics and a dynamical process performing progressive network generation, we develop an analytic approach that accurately captures the dynamical interaction between epidemics on overlay networks. The formalism allows for overlay networks with arbitrary joint degree distribution and overlap. To illustrate the versatility of our approach, we consider a hypothetical delayed intervention scenario in which an immunizing agent is disseminated in a host population to hinder the propagation of an undesirable agent (e.g. the spread of preventive information in the context of an emerging infectious disease).

physics.soc-ph↗

arXiv2010

Propagation dynamics on networks featuring complex topologies

Analytical description of propagation phenomena on random networks has flourished in recent years, yet more complex systems have mainly been studied through numerical means. In this paper, a mean-field description is used to coherently couple the dynamics of the network elements (nodes, vertices, individuals...) on the one hand and their recurrent topological patterns (subgraphs, groups...) on the other hand. In a SIS model of epidemic spread on social networks with community structure, this approach yields a set of ODEs for the time evolution of the system, as well as analytical solutions for the epidemic threshold and equilibria. The results obtained are in good agreement with numerical simulations and reproduce random networks behavior in the appropriate limits which highlights the influence of topology on the processes. Finally, it is demonstrated that our model predicts higher epidemic thresholds for clustered structures than for equivalent random topologies in the case of networks with zero degree correlation.

cond-mat.stat-mech↗

arXiv2010

Adaptive networks: coevolution of disease and topology

Adaptive networks have been recently introduced in the context of disease propagation on complex networks. They account for the mutual interaction between the network topology and the states of the nodes. Until now, existing models have been analyzed using low-complexity analytic formalisms, revealing nevertheless some novel dynamical features. However, current methods have failed to reproduce with accuracy the simultaneous time evolution of the disease and the underlying network topology. In the framework of the adaptive SIS model of Gross et al. [Phys. Rev. Lett. 96, 208701 (2006)], we introduce an improved compartmental formalism able to handle this coevolutionary task successfully. With this approach, we analyze the interplay and outcomes of both dynamical elements, process and structure, on adaptive networks featuring different degree distributions at the initial stage.

cond-mat.stat-mech↗

arXiv2009

Heterogeneous Bond Percolation on Multitype Networks with an Application to Epidemic Dynamics

Considerable attention has been paid, in recent years, to the use of networks in modeling complex real-world systems. Among the many dynamical processes involving networks, propagation processes -- in which final state can be obtained by studying the underlying network percolation properties -- have raised formidable interest. In this paper, we present a bond percolation model of multitype networks with an arbitrary joint degree distribution that allows heterogeneity in the edge occupation probability. As previously demonstrated, the multitype approach allows many non-trivial mixing patterns such as assortativity and clustering between nodes. We derive a number of useful statistical properties of multitype networks as well as a general phase transition criterion. We also demonstrate that a number of previous models based on probability generating functions are special cases of the proposed formalism. We further show that the multitype approach, by naturally allowing heterogeneity in the bond occupation probability, overcomes some of the correlation issues encountered by previous models. We illustrate this point in the context of contact network epidemiology.

q-bio.PE↗